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Kim M Keeling

Showing results (1-10 of 33) with videos related to

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Diseases (Basel, Switzerland)|April 4, 2017
Nonsense Suppression as an Approach to Treat Lysosomal Storage DiseasesKim M Keeling
Journal of Molecular Medicine (Berlin, Germany)|June 20, 2002
Clinically relevant aminoglycosides can suppress disease-associated premature stop mutations in the IDUA and P53 cDNAs in a mammalian translation systemKim M Keeling, David M Bedwell
Elife|March 24, 2020
Finding sense in the contextKim M Keeling, David M Bedwell
Wiley Interdisciplinary Reviews. RNA|October 7, 2011
Suppression of nonsense mutations as a therapeutic approach to treat genetic diseasesKim M Keeling, David M Bedwell
European Journal of Medicinal Chemistry|June 9, 2020
Pharmacological approaches for targeting cystic fibrosis nonsense mutationsJyoti Sharma, Kim M Keeling, Steven M Rowe
Eukaryotic Cell|August 10, 2006
Eukaryotic release factor 1 phosphorylation by CK2 protein kinase is dynamic but has little effect on the efficiency of translation termination in Saccharomyces cerevisiaeAdam K Kallmeyer, Kim M Keeling, David M Bedwell
Annual Review of Genomics and Human Genetics|April 30, 2014
Therapeutics based on stop codon readthroughKim M Keeling, Xiaojiao Xue, Gwen Gunn, et al.
Critical Reviews in Biochemistry and Molecular Biology|June 8, 2012
Suppression of premature termination codons as a therapeutic approachKim M Keeling, Dan Wang, Sara E Conard, et al.
Molecular and Cellular Biology|July 1, 2006
Tpa1p is part of an mRNP complex that influences translation termination, mRNA deadenylation, and mRNA turnover in Saccharomyces cerevisiaeKim M Keeling, Joe Salas-Marco, Lev Z Osherovich, et al.
The Journal of Biological Chemistry|January 13, 2009
Poly-L-aspartic acid enhances and prolongs gentamicin-mediated suppression of the CFTR-G542X mutation in a cystic fibrosis mouse modelMing Du, Kim M Keeling, Liming Fan, et al.
Pageof 4

Showing results (1-10 of 33) with videos related to

Sort By:
Pageof 4
Diseases (Basel, Switzerland)|April 4, 2017
Nonsense Suppression as an Approach to Treat Lysosomal Storage DiseasesKim M Keeling
Journal of Molecular Medicine (Berlin, Germany)|June 20, 2002
Clinically relevant aminoglycosides can suppress disease-associated premature stop mutations in the IDUA and P53 cDNAs in a mammalian translation systemKim M Keeling, David M Bedwell
Elife|March 24, 2020
Finding sense in the contextKim M Keeling, David M Bedwell
Wiley Interdisciplinary Reviews. RNA|October 7, 2011
Suppression of nonsense mutations as a therapeutic approach to treat genetic diseasesKim M Keeling, David M Bedwell
European Journal of Medicinal Chemistry|June 9, 2020
Pharmacological approaches for targeting cystic fibrosis nonsense mutationsJyoti Sharma, Kim M Keeling, Steven M Rowe
Eukaryotic Cell|August 10, 2006
Eukaryotic release factor 1 phosphorylation by CK2 protein kinase is dynamic but has little effect on the efficiency of translation termination in Saccharomyces cerevisiaeAdam K Kallmeyer, Kim M Keeling, David M Bedwell
Annual Review of Genomics and Human Genetics|April 30, 2014
Therapeutics based on stop codon readthroughKim M Keeling, Xiaojiao Xue, Gwen Gunn, et al.
Critical Reviews in Biochemistry and Molecular Biology|June 8, 2012
Suppression of premature termination codons as a therapeutic approachKim M Keeling, Dan Wang, Sara E Conard, et al.
Molecular and Cellular Biology|July 1, 2006
Tpa1p is part of an mRNP complex that influences translation termination, mRNA deadenylation, and mRNA turnover in Saccharomyces cerevisiaeKim M Keeling, Joe Salas-Marco, Lev Z Osherovich, et al.
The Journal of Biological Chemistry|January 13, 2009
Poly-L-aspartic acid enhances and prolongs gentamicin-mediated suppression of the CFTR-G542X mutation in a cystic fibrosis mouse modelMing Du, Kim M Keeling, Liming Fan, et al.
Pageof 4