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Journal of Cellular and Molecular Medicine
|
August 5, 2009
Calcium in the initiation, progression and as an effector of Alzheimer's disease pathology
Kim N Green
Journal of Neurochemistry
|
June 18, 2002
Divergent pathways account for two distinct effects of amyloid beta peptides on exocytosis and Ca(2+) currents: involvement of ROS and NF-kappaB
Kim N Green, Chris Peers
Cold Spring Harbor Perspectives in Medicine
|
September 25, 2012
Animal models of Alzheimer disease
Frank M LaFerla, Kim N Green
Autophagy
|
April 5, 2011
Presenilins mediate efficient proteolysis via the autophagosome-lysosome system
Kara M Neely, Kim N Green
Neuron
|
August 1, 2008
Linking calcium to Abeta and Alzheimer's disease
Kim N Green, Frank M LaFerla
Brain, Behavior, and Immunity
|
July 11, 2016
Inflammation in Alzheimer's disease: Lessons learned from microglia-depletion models
Elizabeth E Spangenberg, Kim N Green
The Journal of Physiology
|
June 18, 2002
Hypoxia potentiates exocytosis and Ca2+ channels in PC12 cells via increased amyloid beta peptide formation and reactive oxygen species generation
Kim N Green, John P Boyle, Chris Peers
Nature Reviews. Neuroscience
|
June 7, 2007
Intracellular amyloid-beta in Alzheimer's disease
Frank M LaFerla, Kim N Green, Salvatore Oddo
Plos One
|
August 8, 2012
Genetic knockdown of brain-derived neurotrophic factor in 3xTg-AD mice does not alter Aβ or tau pathology
Nicholas A Castello, Kim N Green, Frank M LaFerla
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience
|
March 19, 2011
Presenilin is necessary for efficient proteolysis through the autophagy-lysosome system in a γ-secretase-independent manner
Kara M Neely, Kim N Green, Frank M LaFerla
Page
of 13
Search research articles
Search
Showing results (1-10 of 121) with videos related to
Sort By:
Page
of 13
Journal of Cellular and Molecular Medicine
|
August 5, 2009
Calcium in the initiation, progression and as an effector of Alzheimer's disease pathology
Kim N Green
Journal of Neurochemistry
|
June 18, 2002
Divergent pathways account for two distinct effects of amyloid beta peptides on exocytosis and Ca(2+) currents: involvement of ROS and NF-kappaB
Kim N Green, Chris Peers
Cold Spring Harbor Perspectives in Medicine
|
September 25, 2012
Animal models of Alzheimer disease
Frank M LaFerla, Kim N Green
Autophagy
|
April 5, 2011
Presenilins mediate efficient proteolysis via the autophagosome-lysosome system
Kara M Neely, Kim N Green
Neuron
|
August 1, 2008
Linking calcium to Abeta and Alzheimer's disease
Kim N Green, Frank M LaFerla
Brain, Behavior, and Immunity
|
July 11, 2016
Inflammation in Alzheimer's disease: Lessons learned from microglia-depletion models
Elizabeth E Spangenberg, Kim N Green
The Journal of Physiology
|
June 18, 2002
Hypoxia potentiates exocytosis and Ca2+ channels in PC12 cells via increased amyloid beta peptide formation and reactive oxygen species generation
Kim N Green, John P Boyle, Chris Peers
Nature Reviews. Neuroscience
|
June 7, 2007
Intracellular amyloid-beta in Alzheimer's disease
Frank M LaFerla, Kim N Green, Salvatore Oddo
Plos One
|
August 8, 2012
Genetic knockdown of brain-derived neurotrophic factor in 3xTg-AD mice does not alter Aβ or tau pathology
Nicholas A Castello, Kim N Green, Frank M LaFerla
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience
|
March 19, 2011
Presenilin is necessary for efficient proteolysis through the autophagy-lysosome system in a γ-secretase-independent manner
Kara M Neely, Kim N Green, Frank M LaFerla
Page
of 13