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The Journal of Experimental Medicine|October 19, 2005
Innate immunity against Francisella tularensis is dependent on the ASC/caspase-1 axisSanjeev Mariathasan, David S Weiss, Vishva M Dixit, et al.Cell Death and Differentiation|December 11, 2025
cFLIP suppresses caspase-1- and MLKL-independent perinatal lethality driven by auto-processing impaired caspase-8 D387AKim Newton, Katherine E Wickliffe, Allie Maltzman, et al.The Journal of Biological Chemistry|November 26, 2003
Rip2 participates in Bcl10 signaling and T-cell receptor-mediated NF-kappaB activationAstrid A Ruefli-Brasse, Wyne P Lee, Stephen Hurst, et al.Proceedings of the National Academy of Sciences of the United States of America|May 12, 2010
Absent in melanoma 2 is required for innate immune recognition of Francisella tularensisJonathan W Jones, Nobuhiko Kayagaki, Petr Broz, et al.Nature|November 15, 2019
Activity of caspase-8 determines plasticity between cell death pathwaysKim Newton, Katherine E Wickliffe, Allie Maltzman, et al.The Journal of Biological Chemistry|August 8, 2006
The inhibitor of apoptosis protein fusion c-IAP2.MALT1 stimulates NF-kappaB activation independently of TRAF1 AND TRAF2Eugene Varfolomeev, Sarah M Wayson, Vishva M Dixit, et al.The Journal of Biological Chemistry|December 6, 2001
Identification of a novel homotypic interaction motif required for the phosphorylation of receptor-interacting protein (RIP) by RIP3Xiaoqing Sun, Jianping Yin, Melissa A Starovasnik, et al.Immunity|February 22, 2003
Loss of TACI causes fatal lymphoproliferation and autoimmunity, establishing TACI as an inhibitory BLyS receptorDhaya Seshasayee, Patricia Valdez, Minhong Yan, et al.Journal of Molecular Biology|January 19, 2016
Structural Analysis and Optimization of Context-Independent Anti-Hypusine AntibodiesQianting Zhai, Meng He, Aimin Song, et al.The Journal of Biological Chemistry|February 3, 2004
MALT1/paracaspase is a signaling component downstream of CARMA1 and mediates T cell receptor-induced NF-kappaB activationTuanjie Che, Yun You, Donghai Wang, et al.Pageof 15