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Frontiers in Physiology|July 27, 2012
Genetic inactivation of mitochondria-targeted redox enzyme p66ShcA preserves neuronal viability and mitochondrial integrity in response to oxidative challengesKimmy Su, Dennis Bourdette, Michael ForteFrontiers in Physiology|July 31, 2013
Mitochondrial dysfunction and neurodegeneration in multiple sclerosisKimmy Su, Dennis Bourdette, Michael ForteThe European Journal of Neuroscience|July 5, 2011
Mitochondrial calcium and its regulation in neurodegeneration induced by oxidative stressAnna G Barsukova, Dennis Bourdette, Michael ForteThe Journal of Neuroscience : the Official Journal of the Society for Neuroscience|August 31, 2012
Focal increases of axoplasmic Ca2+, aggregation of sodium-calcium exchanger, N-type Ca2+ channel, and actin define the sites of spheroids in axons undergoing oxidative stressAnna G Barsukova, Michael Forte, Dennis BourdetteCurrent Neurology and Neuroscience Reports|August 12, 2009
Axonal degeneration in multiple sclerosis: the mitochondrial hypothesisKimmy G Su, Gary Banker, Dennis Bourdette, et al.The European Journal of Neuroscience|January 25, 2011
Activation of the mitochondrial permeability transition pore modulates Ca2+ responses to physiological stimuli in adult neuronsAnna Barsukova, Alexander Komarov, György Hajnóczky, et al.Neurology. Clinical Practice|November 7, 2013
Postmarketing adverse drug reactions: A duty to report?Eran Klein, Dennis BourdetteCurrent Neurology and Neuroscience Reports|July 5, 2012
New disease-modifying therapies and new challenges for MSVijayshree Yadav, Dennis BourdetteCurrent Neurology and Neuroscience Reports|April 26, 2006
Complementary and alternative medicine: is there a role in multiple sclerosis?Vijayshree Yadav, Dennis BourdetteCurrent Neurology and Neuroscience Reports|July 13, 2011
The radiologically isolated syndrome: look (again) before you treatRebecca Spain, Dennis BourdettePageof 14