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Kekkaku : [Tuberculosis]|April 21, 2012
[Recent advances in tuberculosis immunity]Kiyoko S Akagawa
International Journal of Hematology|July 26, 2002
Functional heterogeneity of colony-stimulating factor-induced human monocyte-derived macrophagesKiyoko S Akagawa
The Journal of Biological Chemistry|October 6, 2005
Catalase plays a critical role in the CSF-independent survival of human macrophages via regulation of the expression of BCL-2 familyIwao Komuro, Tomoyoshi Yasuda, Aikichi Iwamoto, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 23, 2008
Erythromycin derivatives inhibit HIV-1 replication in macrophages through modulation of MAPK activity to induce small isoforms of C/EBPbetaIwao Komuro, Toshiaki Sunazuka, Kiyoko S Akagawa, et al.
Journal of Leukocyte Biology|June 7, 2002
Eicosapentaenoic acid inhibits CSF-induced human monocyte survival and maturation into macrophage through the stimulation of H2O2 productionSachiyo Terada, Mari Takizawa, Shigeru Yamamoto, et al.
Respirology (Carlton, Vic.)|January 21, 2006
Functional heterogeneity of colony-stimulating factor-induced human monocyte-derived macrophagesKiyoko S Akagawa, Iwao Komuro, Hiroko Kanazawa, et al.
Immunology Letters|August 1, 2009
Expression of Th2-skewed pathology mediators in monocyte-derived type 2 of dendritic cells (DC2)Mitsumi Hata, Seiji Takahara, Hidetoshi Tsuzaki, et al.
Bioorganic & Medicinal Chemistry Letters|April 29, 2011
Novel 12-membered non-antibiotic macrolides from erythromycin A; EM900 series as novel leads for anti-inflammatory and/or immunomodulatory agentsAkihiro Sugawara, Akito Sueki, Tomoyasu Hirose, et al.
The Journal of Antibiotics|October 1, 2015
Non-antibiotic 12-membered macrolides: design, synthesis and biological evaluation in a cigarette-smoking modelAkihiro Sugawara, Hideaki Shima, Akito Sueki, et al.
Journal of Immunology (Baltimore, Md. : 1950)|November 8, 2005
Abnormally differentiated subsets of intestinal macrophage play a key role in Th1-dominant chronic colitis through excess production of IL-12 and IL-23 in response to bacteriaNobuhiko Kamada, Tadakazu Hisamatsu, Susumu Okamoto, et al.
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