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Journal of Biochemistry|June 16, 2007
Multiple mechanisms and functions of maf transcription factors in the regulation of tissue-specific genesKohsuke KataokaJournal of Molecular Endocrinology|July 5, 2021
MafA, NeuroD1, and HNF1β synergistically activate the Slc2a2 (Glut2) gene in β-cellsYuka Ono, Kohsuke KataokaJournal of Molecular Endocrinology|August 27, 2024
CREB activates the MafA promoter through proximal E-boxes and a CCAAT motif in pancreatic β-cellsYuki Aida, Kohsuke KataokaJournal of Molecular Endocrinology|December 17, 2024
Glucocorticoids reduce Slc2a2 (GLUT2) gene expression through HNF1 in pancreatic β-cellsYuka Ono, Kohsuke KataokaJournal of Molecular Endocrinology|October 16, 2019
PIASy is a SUMOylation-independent negative regulator of the insulin transactivator MafASuzuka Onishi, Kohsuke KataokaThe Journal of Biological Chemistry|January 20, 2018
Glucose regulates MafA transcription factor abundance and insulin gene expression by inhibiting AMP-activated protein kinase in pancreatic β-cellsRyo Iwaoka, Kohsuke KataokaOncogene|September 13, 2003
MafA has strong cell transforming ability but is a weak transactivatorMakoto Nishizawa, Kohsuke Kataoka, Peter K VogtMolecular and Cellular Endocrinology|April 29, 2015
Gata3 cooperates with Gcm2 and MafB to activate parathyroid hormone gene expression by interacting with SP1Song-Iee Han, Yukino Tsunekage, Kohsuke KataokaEndocrine Journal|September 6, 2007
Roles and regulation of transcription factor MafA in islet beta-cellsShinsaku Aramata, Song-Iee Han, Kohsuke KataokaThe Journal of Biological Chemistry|February 1, 2011
ATF2 interacts with beta-cell-enriched transcription factors, MafA, Pdx1, and beta2, and activates insulin gene transcriptionSong-iee Han, Kunio Yasuda, Kohsuke KataokaPageof 4