Kristen E Hurov - JoVE Visualize

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Genes & Development|September 3, 2010

A genetic screen identifies the Triple T complex required for DNA damage signaling and ATM and ATR stabilityKristen E Hurov, Cecilia Cotta-Ramusino, Stephen J Elledge

Proceedings of the National Academy of Sciences of the United States of America|March 11, 2009

Response of small intestinal epithelial cells to acute disruption of cell division through CDC25 deletionGwanghee Lee, Lynn S White, Kristen E Hurov, et al.

Cell|August 27, 2005

A role for proapoptotic BID in the DNA-damage responseSandra S Zinkel, Kristen E Hurov, Christy Ong, et al.

Molecular Cell|August 4, 2006

Rad17 phosphorylation is required for claspin recruitment and Chk1 activation in response to replication stressXin Wang, Lee Zou, Tao Lu, et al.

Proceedings of the National Academy of Sciences of the United States of America|October 13, 2010

A chromatin localization screen reveals poly (ADP ribose)-regulated recruitment of the repressive polycomb and NuRD complexes to sites of DNA damageDanny M Chou, Britt Adamson, Noah E Dephoure, et al.

Cell|April 7, 2007

Identification of the FANCI protein, a monoubiquitinated FANCD2 paralog required for DNA repairAgata Smogorzewska, Shuhei Matsuoka, Patrizia Vinciguerra, et al.

Science (New York, N.Y.)|May 26, 2007

ATM and ATR substrate analysis reveals extensive protein networks responsive to DNA damageShuhei Matsuoka, Bryan A Ballif, Agata Smogorzewska, et al.

Cancer Research|August 28, 2013

Integrative radiogenomic profiling of squamous cell lung cancerMohamed E Abazeed, Drew J Adams, Kristen E Hurov, et al.

Cancer Research|April 22, 2014

Inhibiting Tankyrases sensitizes KRAS-mutant cancer cells to MEK inhibitors via FGFR2 feedback signalingMarie Schoumacher, Kristen E Hurov, Joseph Lehár, et al.

Cell|July 29, 2017

Project DRIVE: A Compendium of Cancer Dependencies and Synthetic Lethal Relationships Uncovered by Large-Scale, Deep RNAi ScreeningE Robert McDonald, Antoine de Weck, Michael R Schlabach, et al.

Pageof 1

Showing results (1-10 of 10) with videos related to

Sort By:

Pageof 1

Genes & Development|September 3, 2010

A genetic screen identifies the Triple T complex required for DNA damage signaling and ATM and ATR stabilityKristen E Hurov, Cecilia Cotta-Ramusino, Stephen J Elledge

Proceedings of the National Academy of Sciences of the United States of America|March 11, 2009

Response of small intestinal epithelial cells to acute disruption of cell division through CDC25 deletionGwanghee Lee, Lynn S White, Kristen E Hurov, et al.

Cell|August 27, 2005

A role for proapoptotic BID in the DNA-damage responseSandra S Zinkel, Kristen E Hurov, Christy Ong, et al.

Molecular Cell|August 4, 2006

Rad17 phosphorylation is required for claspin recruitment and Chk1 activation in response to replication stressXin Wang, Lee Zou, Tao Lu, et al.

Proceedings of the National Academy of Sciences of the United States of America|October 13, 2010

A chromatin localization screen reveals poly (ADP ribose)-regulated recruitment of the repressive polycomb and NuRD complexes to sites of DNA damageDanny M Chou, Britt Adamson, Noah E Dephoure, et al.

Cell|April 7, 2007

Identification of the FANCI protein, a monoubiquitinated FANCD2 paralog required for DNA repairAgata Smogorzewska, Shuhei Matsuoka, Patrizia Vinciguerra, et al.

Science (New York, N.Y.)|May 26, 2007

ATM and ATR substrate analysis reveals extensive protein networks responsive to DNA damageShuhei Matsuoka, Bryan A Ballif, Agata Smogorzewska, et al.

Cancer Research|August 28, 2013

Integrative radiogenomic profiling of squamous cell lung cancerMohamed E Abazeed, Drew J Adams, Kristen E Hurov, et al.

Cancer Research|April 22, 2014

Inhibiting Tankyrases sensitizes KRAS-mutant cancer cells to MEK inhibitors via FGFR2 feedback signalingMarie Schoumacher, Kristen E Hurov, Joseph Lehár, et al.

Cell|July 29, 2017

Project DRIVE: A Compendium of Cancer Dependencies and Synthetic Lethal Relationships Uncovered by Large-Scale, Deep RNAi ScreeningE Robert McDonald, Antoine de Weck, Michael R Schlabach, et al.

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