Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Kuo Du

Showing results (21-30 of 48) with videos related to

Pageof 5
Sort By:
Toxicology and Applied Pharmacology|September 28, 2013
The gap junction inhibitor 2-aminoethoxy-diphenyl-borate protects against acetaminophen hepatotoxicity by inhibiting cytochrome P450 enzymes and c-jun N-terminal kinase activationKuo Du, C David Williams, Mitchell R McGill, et al.
Food and Chemical Toxicology : an International Journal Published for the British Industrial Biological Research Association|April 1, 2017
Corrigendum to "Differential susceptibility to acetaminophen-induced liver injury in sub-strains of C57BL/6 mice: 6N versus 6J" [Food and Chem. Toxicol. 98 (Part B) (2016) 107-118]Luqi Duan, John S Davis, Benjamin L Woolbright, et al.
Food and Chemical Toxicology : an International Journal Published for the British Industrial Biological Research Association|November 5, 2016
Differential susceptibility to acetaminophen-induced liver injury in sub-strains of C57BL/6 mice: 6N versus 6JLuqi Duan, John S Davis, Benjamin L Woolbright, et al.
Liver International : Official Journal of the International Association for the Study of the Liver|May 15, 2021
Inhibiting xCT/SLC7A11 induces ferroptosis of myofibroblastic hepatic stellate cells but exacerbates chronic liver injuryKuo Du, Seh Hoon Oh, Rajesh K Dutta, et al.
Archives of Toxicology|October 17, 2018
Mito-tempo protects against acute liver injury but induces limited secondary apoptosis during the late phase of acetaminophen hepatotoxicityKuo Du, Anup Ramachandran, James L Weemhoff, et al.
Journal of Hepatology|May 7, 2016
Removal of acetaminophen protein adducts by autophagy protects against acetaminophen-induced liver injury in miceHong-Min Ni, Mitchell R McGill, Xiaojuan Chao, et al.
Toxicology and Applied Pharmacology|October 4, 2015
Mitochondrial protein adducts formation and mitochondrial dysfunction during N-acetyl-m-aminophenol (AMAP)-induced hepatotoxicity in primary human hepatocytesYuchao Xie, Mitchell R McGill, Kuo Du, et al.
Food and Chemical Toxicology : an International Journal Published for the British Industrial Biological Research Association|August 23, 2017
Induction of mitochondrial biogenesis protects against acetaminophen hepatotoxicityKuo Du, Anup Ramachandran, Mitchell R McGill, et al.
Toxicological Sciences : an Official Journal of the Society of Toxicology|January 27, 2017
Dual Role of Epidermal Growth Factor Receptor in Liver Injury and Regeneration after Acetaminophen Overdose in MiceBharat Bhushan, Hemantkumar Chavan, Prachi Borude, et al.
Nature Aging|June 25, 2024
Aging promotes metabolic dysfunction-associated steatotic liver disease by inducing ferroptotic stressKuo Du, Liuyang Wang, Ji Hye Jun, et al.
Pageof 5

Showing results (21-30 of 48) with videos related to

Sort By:
Pageof 5
Toxicology and Applied Pharmacology|September 28, 2013
The gap junction inhibitor 2-aminoethoxy-diphenyl-borate protects against acetaminophen hepatotoxicity by inhibiting cytochrome P450 enzymes and c-jun N-terminal kinase activationKuo Du, C David Williams, Mitchell R McGill, et al.
Food and Chemical Toxicology : an International Journal Published for the British Industrial Biological Research Association|April 1, 2017
Corrigendum to "Differential susceptibility to acetaminophen-induced liver injury in sub-strains of C57BL/6 mice: 6N versus 6J" [Food and Chem. Toxicol. 98 (Part B) (2016) 107-118]Luqi Duan, John S Davis, Benjamin L Woolbright, et al.
Food and Chemical Toxicology : an International Journal Published for the British Industrial Biological Research Association|November 5, 2016
Differential susceptibility to acetaminophen-induced liver injury in sub-strains of C57BL/6 mice: 6N versus 6JLuqi Duan, John S Davis, Benjamin L Woolbright, et al.
Liver International : Official Journal of the International Association for the Study of the Liver|May 15, 2021
Inhibiting xCT/SLC7A11 induces ferroptosis of myofibroblastic hepatic stellate cells but exacerbates chronic liver injuryKuo Du, Seh Hoon Oh, Rajesh K Dutta, et al.
Archives of Toxicology|October 17, 2018
Mito-tempo protects against acute liver injury but induces limited secondary apoptosis during the late phase of acetaminophen hepatotoxicityKuo Du, Anup Ramachandran, James L Weemhoff, et al.
Journal of Hepatology|May 7, 2016
Removal of acetaminophen protein adducts by autophagy protects against acetaminophen-induced liver injury in miceHong-Min Ni, Mitchell R McGill, Xiaojuan Chao, et al.
Toxicology and Applied Pharmacology|October 4, 2015
Mitochondrial protein adducts formation and mitochondrial dysfunction during N-acetyl-m-aminophenol (AMAP)-induced hepatotoxicity in primary human hepatocytesYuchao Xie, Mitchell R McGill, Kuo Du, et al.
Food and Chemical Toxicology : an International Journal Published for the British Industrial Biological Research Association|August 23, 2017
Induction of mitochondrial biogenesis protects against acetaminophen hepatotoxicityKuo Du, Anup Ramachandran, Mitchell R McGill, et al.
Toxicological Sciences : an Official Journal of the Society of Toxicology|January 27, 2017
Dual Role of Epidermal Growth Factor Receptor in Liver Injury and Regeneration after Acetaminophen Overdose in MiceBharat Bhushan, Hemantkumar Chavan, Prachi Borude, et al.
Nature Aging|June 25, 2024
Aging promotes metabolic dysfunction-associated steatotic liver disease by inducing ferroptotic stressKuo Du, Liuyang Wang, Ji Hye Jun, et al.
Pageof 5