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Elife|August 4, 2022
Coupling to short linear motifs creates versatile PME-1 activities in PP2A holoenzyme demethylation and inhibitionYitong Li, Vijaya Kumar Balakrishnan, Michael Rowse, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|May 9, 2008
Protein kinase A anchoring via AKAP150 is essential for TRPV1 modulation by forskolin and prostaglandin E2 in mouse sensory neuronsKatrin Schnizler, Leonid P Shutov, Michael J Van Kanegan, et al.
Cell Calcium|March 9, 2025
Mitochondrial Ca<sup>2+</sup> uniporter b (MCUb) regulates neuronal Ca<sup>2+</sup> dynamics and resistance to ischemic strokeTam Nguyen, Zhihong Lin, Nirav Dhanesha, et al.
Cell Reports|June 13, 2025
Aberrant phase separation of two PKA RIβ neurological disorder mutants leads to mechanistically distinct signaling deficitsEmily H Pool, Alexander Glebov-McCloud, Ha Neul Lee, et al.
The Journal of Biological Chemistry|June 10, 2011
A kinase anchor protein 150 (AKAP150)-associated protein kinase A limits dendritic spine densityYuan Lu, Xiang-ming Zha, Eun Young Kim, et al.
The Journal of Biological Chemistry|July 31, 2010
Rab32 modulates apoptosis onset and mitochondria-associated membrane (MAM) propertiesMichael Bui, Susanna Y Gilady, Ross E B Fitzsimmons, et al.
Biochemistry|March 8, 2006
Binding of protein phosphatase 2A to the L-type calcium channel Cav1.2 next to Ser1928, its main PKA site, is critical for Ser1928 dephosphorylationDuane D Hall, Joel A Feekes, Aruni S Arachchige Don, et al.
Science (New York, N.Y.)|June 4, 2021
Extensive pleiotropism and allelic heterogeneity mediate metabolic effects of <i>IRX3</i> and <i>IRX5</i>Débora R Sobreira, Amelia C Joslin, Qi Zhang, et al.
Molecular Metabolism|December 13, 2022
The BBSome regulates mitochondria dynamics and functionDeng-Fu Guo, Ronald A Merrill, Lan Qian, et al.
Cell Death & Disease|April 22, 2020
Loss of AKAP1 triggers Drp1 dephosphorylation-mediated mitochondrial fission and loss in retinal ganglion cellsGenea Edwards, Guy A Perkins, Keun-Young Kim, et al.
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