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Biomolecules|March 25, 2022
Novel Therapeutic Target for Prevention of Neurodegenerative Diseases: Modulation of Neuroinflammation with Sig-1R LigandsFerenc Bogár, Lívia Fülöp, Botond PenkeMolecules (Basel, Switzerland)|October 11, 2017
β-Amyloid and the Pathomechanisms of Alzheimer's Disease: A Comprehensive ViewBotond Penke, Ferenc Bogár, Lívia FülöpCurrent Protein & Peptide Science|November 3, 2015
Protein Folding and Misfolding, Endoplasmic Reticulum Stress in Neurodegenerative Diseases: in Trace of Novel Drug TargetsBotond Penke, Ferenc Bogár, Lívia FülöpInternational Journal of Molecular Sciences|October 16, 2024
Mica Lattice Orientation of Epitaxially Grown Amyloid β25-35 FibrilsGyörgy G Ferenczy, Ünige Murvai, Lívia Fülöp, et al.Current Protein & Peptide Science|January 4, 2019
Key Peptides and Proteins in Alzheimer's DiseaseBotond Penke, Ferenc Bogár, Gábor Paragi, et al.Biochemical and Biophysical Research Communications|October 7, 2004
Beta-amyloid-derived pentapeptide RIIGLa inhibits Abeta(1-42) aggregation and toxicityLívia Fülöp, Márta Zarándi, Zsolt Datki, et al.Chemistry (Weinheim an Der Bergstrasse, Germany)|March 26, 2014
Exploiting aromatic interactions for β-peptide foldamer helix stabilization: a significant design elementIstván M Mándity, Antonella Monsignori, Lívia Fülöp, et al.Neural Plasticity|October 3, 2014
Abeta(1-42) enhances neuronal excitability in the CA1 via NR2B subunit-containing NMDA receptorsEdina Varga, Gábor Juhász, Zsolt Bozsó, et al.Biomed Research International|July 23, 2014
Simultaneous changes of spatial memory and spine density after intrahippocampal administration of fibrillar aβ1-42 to the rat brainEmőke Borbély, János Horváth, Szabina Furdan, et al.Journal of Alzheimer'S Disease : JAD|December 31, 2014
Amyloid-β1-42 Disrupts Synaptic Plasticity by Altering Glutamate Recycling at the SynapseEdina Varga, Gábor Juhász, Zsolt Bozsó, et al.Pageof 6