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Trends in Cardiovascular Medicine|January 15, 2011
The cardiac cell cycle, pocket proteins, and p300L A Kirshenbaum, M D SchneiderThe Journal of Biological Chemistry|April 7, 1995
Adenovirus E1A represses cardiac gene transcription and reactivates DNA synthesis in ventricular myocytes, via alternative pocket protein- and p300-binding domainsL A Kirshenbaum, M D SchneiderDevelopmental Biology|November 1, 1996
Human E2F-1 reactivates cell cycle progression in ventricular myocytes and represses cardiac gene transcriptionL A Kirshenbaum, M Abdellatif, S Chakraborty, et al.Molecular Reproduction and Development|September 1, 1994
Control of cardiac gene transcription by fibroblast growth factorsM D Schneider, L A Kirshenbaum, T Brand, et al.The Journal of Clinical Investigation|July 1, 1993
Highly efficient gene transfer into adult ventricular myocytes by recombinant adenovirusL A Kirshenbaum, W R MacLellan, W Mazur, et al.The Canadian Journal of Cardiology|April 29, 1998
Regulators of apoptosis in the heart: a matter of life and deathL A KirshenbaumMolecular and Cellular Biochemistry|July 1, 1997
Adenovirus mediated-gene transfer into cardiomyocytesL A KirshenbaumClinical and Investigative Medicine. Medecine Clinique Et Experimentale|October 31, 2000
Bcl-2 intersects the NFkappaB signalling pathway and suppresses apoptosis in ventricular myocytesL A KirshenbaumThe Journal of Clinical Investigation|February 12, 1998
Adenoviral delivery of E2F-1 directs cell cycle reentry and p53-independent apoptosis in postmitotic adult myocardium in vivoR Agah, L A Kirshenbaum, M Abdellatif, et al.Apoptosis : an International Journal on Programmed Cell Death|November 25, 2003
Apoptosis in ventricular myocytes: the role of tumor suppressor proteinsK Regula, L A KirshenbaumPageof 10