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The American Journal of Pathology|November 14, 1997
Keratinocytes derived from psoriatic plaques are resistant to apoptosis compared with normal skinT Wrone-Smith, R S Mitra, C B Thompson, et al.
European Journal of Immunology|May 1, 1995
The role of bcl-XL in CD40-mediated rescue from anti-mu-induced apoptosis in WEHI-231 B lymphoma cellsM S Choi, L H Boise, A R Gottschalk, et al.
Molecular and Cellular Biology|February 1, 1994
Activation of the granulocyte-macrophage colony-stimulating factor promoter in T cells requires cooperative binding of Elf-1 and AP-1 transcription factorsC Y Wang, A G Bassuk, L H Boise, et al.
Journal of Virology|October 1, 1992
A novel Ets-related transcription factor, Elf-1, binds to human immunodeficiency virus type 2 regulatory elements that are required for inducible trans activation in T cellsJ M Leiden, C Y Wang, B Petryniak, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 23, 1995
Bclx regulates the survival of double-positive thymocytesA Ma, J C Pena, B Chang, et al.
Journal of Immunological Methods|October 28, 1983
A method for size separation of murine spleen cells using counterflow centrifugationC B Thompson, J J Ryan, D G Sieckmann, et al.
The Journal of Experimental Medicine|September 1, 1996
Limited role of CD28-mediated signals in T helper subset differentiationD R Brown, J M Green, N H Moskowitz, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 9, 1995
bcl-x is expressed in embryonic and postnatal neural tissues and functions to prevent neuronal cell deathM González-García, I García, L Ding, et al.
Molecular and Cellular Biology|September 22, 1999
The BH3 domain of Bcl-x(S) is required for inhibition of the antiapoptotic function of Bcl-x(L)B S Chang, A Kelekar, M H Harris, et al.
The Journal of Biological Chemistry|December 5, 2000
Interleukin-3 withdrawal induces an early increase in mitochondrial membrane potential unrelated to the Bcl-2 family. Roles of intracellular pH, ADP transport, and F(0)F(1)-ATPaseA R Khaled, D A Reynolds, H A Young, et al.
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