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Genetic Epidemiology|January 1, 1989
Modeling the age-of-onset function in segregation analysis: a causal scheme for leprosyL Abel, A Mallet, F Demenais, et al.Genetic Epidemiology|January 1, 1989
Robustness of the unified model to shared environmental effects in the analysis of dichotomous traitsF Demenais, L AbelAmerican Journal of Human Genetics|February 1, 1988
Detection of major genes for susceptibility to leprosy and its subtypes in a Caribbean island: Desirade islandL Abel, F DemenaisGenetic Epidemiology|January 1, 1990
A time-dependent logistic hazard function for modeling variable age of onset in analysis of familial diseasesL Abel, G E BonneyGenetic Epidemiology|January 1, 1995
Complex segregation analysis of familial diseases with variable age of onset: comparison of different methods by a simulation studyL Abel, A Garcia, F DemenaisGenetic Epidemiology|January 1, 1995
How can maximum likelihood methods reveal candidate gene effects on a quantitative trait?M Martinez, L Abel, F DemenaisGenetic Epidemiology|July 22, 1998
Comparison of four sib-pair linkage methods for analyzing sibships with more than two affecteds: interest of the binomial maximum likelihood approachL Abel, A Alcais, A MalletAmerican Journal of Human Genetics|May 1, 1991
Evidence for the segregation of a major gene in human susceptibility/resistance to infection by Schistosoma mansoniL Abel, F Demenais, A Prata, et al.American Journal of Human Genetics|October 1, 1993
An autologistic model for the genetic analysis of familial binary dataL Abel, J L Golmard, A MalletThe American Journal of Tropical Medicine and Hygiene|May 9, 1998
Genetic control of blood infection levels in human malaria: evidence for a complex genetic modelA Garcia, M Cot, J P Chippaux, et al.Pageof 70