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Proceedings of the National Academy of Sciences of the United States of America|May 16, 2007
In vitro erythropoiesis from bone marrow-derived progenitors provides a physiological assay for toxic and mutagenic compoundsJ Shuga, J Zhang, L D Samson, et al.Proceedings of the National Academy of Sciences of the United States of America|December 6, 2000
Molecular basis for discriminating between normal and damaged bases by the human alkyladenine glycosylase, AAGA Y Lau, M D Wyatt, B J Glassner, et al.Bioessays : News and Reviews in Molecular, Cellular and Developmental Biology|August 10, 1999
3-methyladenine DNA glycosylases: structure, function, and biological importanceM D Wyatt, J M Allan, A Y Lau, et al.Proceedings of the National Academy of Sciences of the United States of America|August 26, 1998
Generation of a strong mutator phenotype in yeast by imbalanced base excision repairB J Glassner, L J Rasmussen, M T Najarian, et al.Cancer Research|September 10, 1998
Mammalian 3-methyladenine DNA glycosylase protects against the toxicity and clastogenicity of certain chemotherapeutic DNA cross-linking agentsJ M Allan, B P Engelward, A J Dreslin, et al.Carcinogenesis|February 1, 1993
Cloning and characterization of a mouse 3-methyladenine/7-methyl-guanine/3-methylguanine DNA glycosylase cDNA whose gene maps to chromosome 11B P Engelward, M S Boosalis, B J Chen, et al.The Journal of Biological Chemistry|March 28, 1998
A chemical and genetic approach together define the biological consequences of 3-methyladenine lesions in the mammalian genomeB P Engelward, J M Allan, A J Dreslin, et al.DNA Repair|January 24, 2009
DNA repair modulates the vulnerability of the developing brain to alkylating agentsG E Kisby, A Olivas, T Park, et al.Current Drug Metabolism|December 29, 2005
A microscale in vitro physiological model of the liver: predictive screens for drug metabolism and enzyme inductionA Sivaraman, J K Leach, S Townsend, et al.Proceedings of the National Academy of Sciences of the United States of America|December 16, 1997
Base excision repair deficient mice lacking the Aag alkyladenine DNA glycosylaseB P Engelward, G Weeda, M D Wyatt, et al.Pageof 3