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Biochemical Society Transactions|October 21, 2006
Do voltage-gated calcium channel alpha2delta subunits require proteolytic processing into alpha2 and delta to be functional?L Douglas, A Davies, J Wratten, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 3, 2005
The metal-ion-dependent adhesion site in the Von Willebrand factor-A domain of alpha2delta subunits is key to trafficking voltage-gated Ca2+ channelsC Cantí, M Nieto-Rostro, I Foucault, et al.
Brain Research|December 2, 1982
Noradrenergic modulation of glutamate release in the cerebellumA C Dolphin
Trends in Neurosciences|January 1, 1996
Facilitation of Ca2+ current in excitable cellsA C Dolphin
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|October 19, 2001
Dominant-negative synthesis suppression of voltage-gated calcium channel Cav2.2 induced by truncated constructsA Raghib, F Bertaso, A Davies, et al.
Biophysical Journal|August 18, 2001
Evidence for two concentration-dependent processes for beta-subunit effects on alpha1B calcium channelsC Cantí, A Davies, N S Berrow, et al.
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