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Showing results (41-50 of 47) with videos related to

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Progress in Clinical and Biological Research|January 1, 1989
DNA repair deficiency for alkylation damage in cells from Alzheimer's disease patientsW G Bradley, R J Polinsky, W W Pendlebury, et al.
Annals of Neurology|December 16, 2000
Frontotemporal dementia with novel tau pathology and a Glu342Val tau mutationC F Lippa, V Zhukareva, T Kawarai, et al.
Annals of Neurology|September 8, 2000
Familial Alzheimer's disease: site of mutation influences clinical phenotypeC F Lippa, J M Swearer, K J Kane, et al.
FEBS Letters|December 31, 1997
Proteolytic processing of presenilin-1 (PS-1) is not associated with Alzheimer's disease with or without PS-1 mutationsM Okochi, K Ishii, M Usami, et al.
The American Journal of Pathology|November 12, 1998
Lewy bodies contain altered alpha-synuclein in brains of many familial Alzheimer's disease patients with mutations in presenilin and amyloid precursor protein genesC F Lippa, H Fujiwara, D M Mann, et al.
Neurobiology of Aging|May 30, 2001
Distinguishable effects of presenilin-1 and APP717 mutations on amyloid plaque depositionK Ishii, C Lippa, T Tomiyama, et al.
Brain Research. Molecular Brain Research|May 29, 1998
Amyloid-beta-protein isoforms in brain of subjects with PS1-linked, beta APP-linked and sporadic Alzheimer diseaseA Tamaoka, P E Fraser, K Ishii, et al.
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Showing results (41-50 of 47) with videos related to

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Pageof 5
You have reached the last page of results.This site can display upto 47 results.
Progress in Clinical and Biological Research|January 1, 1989
DNA repair deficiency for alkylation damage in cells from Alzheimer's disease patientsW G Bradley, R J Polinsky, W W Pendlebury, et al.
Annals of Neurology|December 16, 2000
Frontotemporal dementia with novel tau pathology and a Glu342Val tau mutationC F Lippa, V Zhukareva, T Kawarai, et al.
Annals of Neurology|September 8, 2000
Familial Alzheimer's disease: site of mutation influences clinical phenotypeC F Lippa, J M Swearer, K J Kane, et al.
FEBS Letters|December 31, 1997
Proteolytic processing of presenilin-1 (PS-1) is not associated with Alzheimer's disease with or without PS-1 mutationsM Okochi, K Ishii, M Usami, et al.
The American Journal of Pathology|November 12, 1998
Lewy bodies contain altered alpha-synuclein in brains of many familial Alzheimer's disease patients with mutations in presenilin and amyloid precursor protein genesC F Lippa, H Fujiwara, D M Mann, et al.
Neurobiology of Aging|May 30, 2001
Distinguishable effects of presenilin-1 and APP717 mutations on amyloid plaque depositionK Ishii, C Lippa, T Tomiyama, et al.
Brain Research. Molecular Brain Research|May 29, 1998
Amyloid-beta-protein isoforms in brain of subjects with PS1-linked, beta APP-linked and sporadic Alzheimer diseaseA Tamaoka, P E Fraser, K Ishii, et al.
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