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Novartis Foundation Symposium|March 19, 2005
Skeletal and cardiac muscle defects in a murine model of Emery-Dreifuss muscular dystrophyM J Grattan, C Kondo, J Thurston, et al.
Mathematical Biosciences|September 1, 1991
Conduction in bullfrog atrial strands: simulations of the role of disc and extracellular resistanceC R Murphey, J W Clark, W R Giles, et al.
The American Journal of Physiology|August 1, 1990
A mathematical model of electrophysiological activity in a bullfrog atrial cellR L Rasmusson, J W Clark, W R Giles, et al.
Pflugers Archiv : European Journal of Physiology|June 17, 1999
The isolated working mouse heart: methodological considerationsT S Larsen, D D Belke, R Sas, et al.
Nature|March 24, 1998
Spatiotemporal evolution of ventricular fibrillationF X Witkowski, L J Leon, P A Penkoske, et al.
American Journal of Transplantation : Official Journal of the American Society of Transplantation and the American Society of Transplant Surgeons|March 19, 2009
Blockade of K(ATP) channels reduces endothelial hyperpolarization and leukocyte recruitment upon reperfusion after hypoxiaM Figura, L Chilton, A Liacini, et al.
American Journal of Physiology. Heart and Circulatory Physiology|January 18, 2005
K+ currents regulate the resting membrane potential, proliferation, and contractile responses in ventricular fibroblasts and myofibroblastsL Chilton, S Ohya, D Freed, et al.
Cell|November 11, 1999
Chronic phospholamban-sarcoplasmic reticulum calcium ATPase interaction is the critical calcium cycling defect in dilated cardiomyopathyS Minamisawa, M Hoshijima, G Chu, et al.
Experimental Physiology|May 24, 2018
ATP triggers a robust intracellular [Ca<sup>2+</sup> ]-mediated signalling pathway in human synovial fibroblastsC Kondo, R B Clark, N Al‐Jezani, et al.
Cell|December 19, 2001
A defect in the Kv channel-interacting protein 2 (KChIP2) gene leads to a complete loss of I(to) and confers susceptibility to ventricular tachycardiaH C Kuo, C F Cheng, R B Clark, et al.
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