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Kidney International|September 18, 1997
Hypoxia stimulates proximal tubular cell matrix production via a TGF-beta1-independent mechanismC Orphanides, L G Fine, J T NormanExperimental Nephrology|November 13, 1999
Hypoxia-induced changes in extracellular matrix metabolism in renal cellsJ T Norman, C Orphanides, P Garcia, et al.Experimental Nephrology|April 24, 1999
Progressive renal disease: fibroblasts, extracellular matrix, and integrinsJ T Norman, L G FineJournal of the American Society of Nephrology : JASN|April 1, 1992
Renal growth responses to acute and chronic injury: routes to therapeutic interventionL G Fine, J T NormanKidney International. Supplement|June 1, 2000
Is there a common mechanism for the progression of different types of renal diseases other than proteinuria? Towards the unifying theme of chronic hypoxiaL G Fine, D Bandyopadhay, J T NormanKidney International|January 1, 1996
Pexicrine effects of basement membrane components on paracrine signaling by renal tubular cellsM P Lewis, L G Fine, J T NormanEuropean Journal of Clinical Investigation|May 1, 1993
Mechanisms of tubulo-interstitial injury in progressive renal diseasesL G Fine, A C Ong, J T NormanThe American Journal of Physiology|June 1, 1991
Primary response gene expression in renal hypertrophy and hyperplasia: evidence for different growth initiation processesD A Kujubu, J T Norman, H R Herschman, et al.The American Journal of Physiology|August 1, 1991
Fibroblasts of rabbit kidney in culture. I. Characterization and identification of cell-specific markersH P Rodemann, G A Müller, A Knecht, et al.Journal of the American Society of Nephrology : JASN|November 11, 1992
Stimulation of Na+/H+ exchange is not required for induction of hypertrophy of renal cells in vitroM Mackovic-Basic, L G Fine, J T Norman, et al.Pageof 10