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Brain Research Bulletin|September 1, 1989
Persistent alterations in GABAA receptor binding and function after prenatal lorazepam administration in the chickL G Miller, R B Roy, C L Weill, et al.
Molecular Pharmacology|November 1, 1989
Chronic clonazepam administration decreases gamma-aminobutyric acidA receptor function in cultured cortical neuronsL G Miller, R B Roy, C L Weill
Neurotoxicology|January 1, 1989
Prenatal lorazepam administration is associated with GABAA receptor alterations in late embryonic and mature chicksL G Miller, C L Weill, R B Roy
Brain Research. Developmental Brain Research|December 1, 1988
Lorazepam administration during embryonic development alters GABAA receptor binding and functionL G Miller, C L Weill, R B Roy, et al.
The Journal of Pharmacology and Experimental Therapeutics|January 1, 1989
Dietary choline intake modulates benzodiazepine receptor binding and gamma-aminobutyric acidA receptor function in mouse brainL G Miller, D J Greenblatt, R B Roy, et al.
Biochemical Pharmacology|November 1, 1989
Chronic benzodiazepine administration. IV. Rapid development of tolerance and receptor downregulation associated with alprazolam administrationL G Miller, S Woolverton, D J Greenblatt, et al.
The Journal of Pharmacology and Experimental Therapeutics|March 1, 1989
Chronic benzodiazepine administration. III. Upregulation of gamma-aminobutyric acidA receptor binding and function associated with chronic benzodiazepine antagonist administrationL G Miller, D J Greenblatt, R B Roy, et al.
Brain Research Bulletin|December 1, 1987
Effects of Tyr-MIF-1 and MIF-1 at the GABAA receptor chloride channel siteL G Miller, A J Kastin, R B Roy
Brain Research Bulletin|December 1, 1989
MIF-1 and Tyr-MIF-1 augment muscimol-stimulated chloride uptake in cerebral cortexL G Miller, A J Kastin, R B Roy
Biochemical Pharmacology|September 15, 1990
Augmentation of GABAA receptor function by chronic exposure to GABA-neutral and GABA-negative benzodiazepine ligands in cultured cortical neuronsL G Miller, J Heller, M Lumpkin, et al.
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