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Thrombosis Research|March 1, 1989
Homozygous protein C deficiency with delayed onset of symptoms at 7 to 10 monthsE G Tuddenham, T Takase, A E Thomas, et al.Nucleic Acids Research|November 11, 1994
Haemophilia A: database of nucleotide substitutions, deletions, insertions and rearrangements of the factor VIII gene, second editionE G Tuddenham, R Schwaab, J Seehafer, et al.Blood|May 15, 1996
Activation of factor VII during alimentary lipemia occurs in healthy adults and patients with congenital factor XII or factor XI deficiency, but not in patients with factor IX deficiencyG J Miller, J C Martin, K A Mitropoulos, et al.Nucleic Acids Research|September 25, 1991
Haemophilia A: database of nucleotide substitutions, deletions, insertions and rearrangements of the factor VIII geneE G Tuddenham, D N Cooper, J Gitschier, et al.British Journal of Haematology|November 5, 1997
Factor VIII gene analysis in Japanese CRM-positive and CRM-reduced haemophilia A patients by single-strand conformation polymorphismS Morichika, M Shima, S Kamisue, et al.Biochemistry|November 29, 1994
Surface plasmon resonance studies of the interaction between factor VII and tissue factor. Demonstration of defective tissue factor binding in a variant FVII molecule (FVII-R79Q)D P O'Brien, G Kemball-Cook, A M Hutchinson, et al.Human Molecular Genetics|September 1, 1993
Detection of missense mutations by single-strand conformational polymorphism (SSCP) analysis in five dysfunctional variants of coagulation factor VIIO Takamiya, G Kemball-Cook, D M Martin, et al.Thrombosis and Haemostasis|May 1, 1997
Factor VIII Ise (R2159C) in a patient with mild hemophilia A, an abnormal factor VIII with retention of function but modification of C2 epitopesH Suzuki, M Shima, M Arai, et al.Blood|March 26, 1999
Molecular analysis of the ERGIC-53 gene in 35 families with combined factor V-factor VIII deficiencyM Neerman-Arbez, K M Johnson, M A Morris, et al.Thrombosis and Haemostasis|October 6, 2000
An alloantibody recognizing the FVIII A1 domain in a patient with CRM reduced haemophilia A due to deletion of a large portion of the A1 domain DNA sequenceM Shibata, M Shima, S Morichika, et al.Pageof 14