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Restorative Neurology and Neuroscience
|
April 3, 2003
The difference between mechanisms of kainate and glutamate excitotoxicity in vitro: osmotic lesion versus mitochondrial depolarization
L Kiedrowski
Polish Journal of Pharmacology and Pharmacy
|
March 1, 1991
The distribution of serotonergic 5-HT1A and non-5-HT1A recognition sites along the dorso-ventral axis of the rat hippocampus
L Kiedrowski
Neuroreport
|
December 6, 2001
Repolarization of the plasma membrane shapes NMDA-induced cytosolic [Ca2+ ] transients
L Kiedrowski
Molecular Pharmacology
|
September 25, 1999
Elevated extracellular K(+) concentrations inhibit N-methyl-D-aspartate-induced Ca(2+) influx and excitotoxicity
L Kiedrowski
Molecular Pharmacology
|
August 27, 1999
N-methyl-D-aspartate excitotoxicity: relationships among plasma membrane potential, Na(+)/Ca(2+) exchange, mitochondrial Ca(2+) overload, and cytoplasmic concentrations of Ca(2+), H(+), and K(+)
L Kiedrowski
Molecular Pharmacology
|
January 1, 1995
Glutamate-induced destabilization of intracellular calcium concentration homeostasis in cultured cerebellar granule cells: role of mitochondria in calcium buffering
L Kiedrowski, E Costa
Neuroreport
|
February 24, 2001
Kainate-induced K+ efflux and plasma membrane depolarization in cultured cerebellar granule cells
L Kiedrowski, J M Mienville
Molecular Pharmacology
|
April 1, 1992
Sodium nitroprusside inhibits N-methyl-D-aspartate-evoked calcium influx via a nitric oxide- and cGMP-independent mechanism
L Kiedrowski, E Costa, J T Wroblewski
Journal of Neurochemistry
|
January 1, 1992
Glutamate receptor agonists stimulate nitric oxide synthase in primary cultures of cerebellar granule cells
L Kiedrowski, E Costa, J T Wroblewski
Neuroscience Letters
|
January 20, 1992
In vitro interaction between cerebellar astrocytes and granule cells: a putative role for nitric oxide
L Kiedrowski, E Costa, J T Wroblewski
Page
of 2
Search research articles
Search
Showing results (1-10 of 18) with videos related to
Sort By:
Page
of 2
Restorative Neurology and Neuroscience
|
April 3, 2003
The difference between mechanisms of kainate and glutamate excitotoxicity in vitro: osmotic lesion versus mitochondrial depolarization
L Kiedrowski
Polish Journal of Pharmacology and Pharmacy
|
March 1, 1991
The distribution of serotonergic 5-HT1A and non-5-HT1A recognition sites along the dorso-ventral axis of the rat hippocampus
L Kiedrowski
Neuroreport
|
December 6, 2001
Repolarization of the plasma membrane shapes NMDA-induced cytosolic [Ca2+ ] transients
L Kiedrowski
Molecular Pharmacology
|
September 25, 1999
Elevated extracellular K(+) concentrations inhibit N-methyl-D-aspartate-induced Ca(2+) influx and excitotoxicity
L Kiedrowski
Molecular Pharmacology
|
August 27, 1999
N-methyl-D-aspartate excitotoxicity: relationships among plasma membrane potential, Na(+)/Ca(2+) exchange, mitochondrial Ca(2+) overload, and cytoplasmic concentrations of Ca(2+), H(+), and K(+)
L Kiedrowski
Molecular Pharmacology
|
January 1, 1995
Glutamate-induced destabilization of intracellular calcium concentration homeostasis in cultured cerebellar granule cells: role of mitochondria in calcium buffering
L Kiedrowski, E Costa
Neuroreport
|
February 24, 2001
Kainate-induced K+ efflux and plasma membrane depolarization in cultured cerebellar granule cells
L Kiedrowski, J M Mienville
Molecular Pharmacology
|
April 1, 1992
Sodium nitroprusside inhibits N-methyl-D-aspartate-evoked calcium influx via a nitric oxide- and cGMP-independent mechanism
L Kiedrowski, E Costa, J T Wroblewski
Journal of Neurochemistry
|
January 1, 1992
Glutamate receptor agonists stimulate nitric oxide synthase in primary cultures of cerebellar granule cells
L Kiedrowski, E Costa, J T Wroblewski
Neuroscience Letters
|
January 20, 1992
In vitro interaction between cerebellar astrocytes and granule cells: a putative role for nitric oxide
L Kiedrowski, E Costa, J T Wroblewski
Page
of 2