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Journal of Neural Transmission. Parkinson'S Disease and Dementia Section|January 1, 1991
Metals and trace elements in plasma and cerebrospinal fluid in normal aging and Alzheimer's diseaseH Basun, L G Forssell, L Wetterberg, et al.Journal of Cellular and Molecular Medicine|August 10, 2002
APOE polymorphism and clinical duration determine regional neuropathology in Swedish APP(670, 671) mutation carriers: implications for late-onset Alzheimer's diseaseN Bogdanovic, Elizabeth Corder, L Lannfelt, et al.Neuroscience Letters|August 23, 1996
Increasing cerebrospinal fluid tau levels in a subgroup of Alzheimer patients with apolipoprotein E allele epsilon 4 during 14 months follow-upM Blomberg, M Jensen, H Basun, et al.Journal of Neural Transmission. Supplementum|November 29, 2002
Conversion from preclinical to dinical stage of Alzheimer's disease as shown by decline of cognitive function in carriers of the Swedish APP-mutationO Almkvist, K Axelman, H Basun, et al.Neuroscience Letters|January 22, 2000
Tau gene polymorphisms and apolipoprotein E epsilon4 may interact to increase risk for Alzheimer's diseaseL Lilius, S Froelich Fabre, H Basun, et al.Dementia and Geriatric Cognitive Disorders|October 26, 2013
High tau levels in cerebrospinal fluid predict rapid decline and increased dementia mortality in Alzheimer's diseaseM Degerman Gunnarsson, L Lannfelt, M Ingelsson, et al.Dementia and Geriatric Cognitive Disorders|February 15, 2001
Cerebrospinal fluid tau levels increase with age in healthy individualsM Blomberg, M Jensen, H Basun, et al.Archives of Gerontology and Geriatrics|May 1, 1996
Apolipoprotein E epsilon4 allele, skin vessel reactivity and vascular risk factors in Alzheimer's diseaseA Algotsson, A Nordberg, L Lannfelt, et al.Nature Medicine|August 1, 1995
Decreased alpha-secretase-cleaved amyloid precursor protein as a diagnostic marker for Alzheimer's diseaseL Lannfelt, H Basun, L O Wahlund, et al.Neurochemistry International|July 1, 1994
The significance of the Swedish APP670/671 mutation for the development of Alzheimer's disease amyloidosisJ Johnston, C O'Neill, L Lannfelt, et al.Pageof 69