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The Journal of Clinical Endocrinology and Metabolism|June 18, 1999
Effect of testosterone treatment on bone mineral density in men over 65 years of ageP J Snyder, H Peachey, P Hannoush, et al.
Nature Communications|December 21, 2017
Genome-wide DNA methylation is predictive of outcome in juvenile myelomonocytic leukemiaElliot Stieglitz, Tali Mazor, Adam B Olshen, et al.
Pediatric Blood & Cancer|August 8, 2023
Palbociclib in combination with chemotherapy in pediatric and young adult patients with relapsed/refractory acute lymphoblastic leukemia and lymphoma: A Children's Oncology Group study (AINV18P1)Elizabeth A Raetz, David T Teachey, Charles Minard, et al.
Ejhaem|August 21, 2023
SARS-CoV-2 infections in patients enrolled on the Children's Oncology Group standard-risk B-cell acute lymphoblastic leukemia trial, AALL1731Caitlin W Elgarten, John A Kairalla, Joel C Thompson, et al.
Pediatric Blood & Cancer|June 29, 2019
Sustained remission with azacitidine monotherapy and an aberrant precursor B-lymphoblast population in juvenile myelomonocytic leukemiaSaman K Hashmi, Jyotinder N Punia, Andrea N Marcogliese, et al.
Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|November 3, 2020
International Consensus Definition of DNA Methylation Subgroups in Juvenile Myelomonocytic LeukemiaMaximilian Schönung, Julia Meyer, Peter Nöllke, et al.
Blood Advances|January 4, 2018
Oncogenic role and therapeutic targeting of ABL-class and JAK-STAT activating kinase alterations in Ph-like ALLKathryn G Roberts, Yung-Li Yang, Debbie Payne-Turner, et al.
Blood|June 12, 2012
Aberrant STAT5 and PI3K/mTOR pathway signaling occurs in human CRLF2-rearranged B-precursor acute lymphoblastic leukemiaSarah K Tasian, Michelle Y Doral, Michael J Borowitz, et al.
Blood|April 21, 2017
Suppression of B-cell development genes is key to glucocorticoid efficacy in treatment of acute lymphoblastic leukemiaKarina A Kruth, Mimi Fang, Dawne N Shelton, et al.
Leukemia|October 19, 2007
Synergistic antileukemic effects between ABT-869 and chemotherapy involve downregulation of cell cycle-regulated genes and c-Mos-mediated MAPK pathwayJ Zhou, M Pan, Z Xie, et al.
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