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L M Boxer

Showing results (31-40 of 44) with videos related to

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Nucleic Acids Research|September 2, 1998
The N-terminal domain unique to the long form of the Brn-3a transcription factor is essential to protect neuronal cells from apoptosis and for the activation of Bbcl-2 gene expressionM D Smith, S J Dawson, L M Boxer, et al.
Lancet (London, England)|April 26, 1997
A woman who had a stroke, then a myocardial infarctionC Ciesielski-Carlucci, B K Lee, L M Boxer, et al.
Oncogene|April 21, 2001
Negative regulation of bcl-2 expression by p53 in hematopoietic cellsY Wu, J W Mehew, C A Heckman, et al.
Experimental Hematology|May 30, 2001
Suppression of apoptosis and granulocyte colony-stimulating factor-induced differentiation by an oncogenic form of CblS Sinha, J Jancarik, V Roginskaya, et al.
Oncogene|January 10, 2002
The critical role of the PE21 element in oncostatin M-mediated transcriptional repression of the p53 tumor suppressor gene in breast cancer cellsC Li, T E Ahlborn, K Tokita, et al.
The Journal of Biological Chemistry|June 27, 1998
Bcl-2 transcription from the proximal P2 promoter is activated in neuronal cells by the Brn-3a POU family transcription factorM D Smith, E A Ensor, R S Coffin, et al.
Cell Growth & Differentiation : the Molecular Biology Journal of the American Association for Cancer Research|November 5, 1999
The expression of p53 tumor suppressor gene in breast cancer cells is down-regulated by cytokine oncostatin MJ Liu, C Li, T E Ahlborn, et al.
Leukemia|May 25, 2011
The immunoglobulin heavy chain gene 3' enhancers induce Bcl2 deregulation and lymphomagenesis in murine B cellsH Xiang, E J Noonan, J Wang, et al.
The Journal of Biological Chemistry|February 29, 2000
A-Myb up-regulates Bcl-2 through a Cdx binding site in t(14;18) lymphoma cellsC A Heckman, J W Mehew, G G Ying, et al.
The Journal of Biological Chemistry|September 17, 1999
Insulin-like growth factor-I induces bcl-2 promoter through the transcription factor cAMP-response element-binding proteinS Pugazhenthi, E Miller, C Sable, et al.
Pageof 5

Showing results (31-40 of 44) with videos related to

Sort By:
Pageof 5
Nucleic Acids Research|September 2, 1998
The N-terminal domain unique to the long form of the Brn-3a transcription factor is essential to protect neuronal cells from apoptosis and for the activation of Bbcl-2 gene expressionM D Smith, S J Dawson, L M Boxer, et al.
Lancet (London, England)|April 26, 1997
A woman who had a stroke, then a myocardial infarctionC Ciesielski-Carlucci, B K Lee, L M Boxer, et al.
Oncogene|April 21, 2001
Negative regulation of bcl-2 expression by p53 in hematopoietic cellsY Wu, J W Mehew, C A Heckman, et al.
Experimental Hematology|May 30, 2001
Suppression of apoptosis and granulocyte colony-stimulating factor-induced differentiation by an oncogenic form of CblS Sinha, J Jancarik, V Roginskaya, et al.
Oncogene|January 10, 2002
The critical role of the PE21 element in oncostatin M-mediated transcriptional repression of the p53 tumor suppressor gene in breast cancer cellsC Li, T E Ahlborn, K Tokita, et al.
The Journal of Biological Chemistry|June 27, 1998
Bcl-2 transcription from the proximal P2 promoter is activated in neuronal cells by the Brn-3a POU family transcription factorM D Smith, E A Ensor, R S Coffin, et al.
Cell Growth & Differentiation : the Molecular Biology Journal of the American Association for Cancer Research|November 5, 1999
The expression of p53 tumor suppressor gene in breast cancer cells is down-regulated by cytokine oncostatin MJ Liu, C Li, T E Ahlborn, et al.
Leukemia|May 25, 2011
The immunoglobulin heavy chain gene 3' enhancers induce Bcl2 deregulation and lymphomagenesis in murine B cellsH Xiang, E J Noonan, J Wang, et al.
The Journal of Biological Chemistry|February 29, 2000
A-Myb up-regulates Bcl-2 through a Cdx binding site in t(14;18) lymphoma cellsC A Heckman, J W Mehew, G G Ying, et al.
The Journal of Biological Chemistry|September 17, 1999
Insulin-like growth factor-I induces bcl-2 promoter through the transcription factor cAMP-response element-binding proteinS Pugazhenthi, E Miller, C Sable, et al.
Pageof 5