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Neuroscience
|
October 19, 2000
Amyloid-like inclusions in Huntington's disease
D P McGowan, W van Roon-Mom, H Holloway, et al.
Human Molecular Genetics
|
April 10, 1999
Formation of polyglutamine inclusions in non-CNS tissue
K Sathasivam, C Hobbs, M Turmaine, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience
|
June 24, 2000
Abnormal synaptic plasticity and impaired spatial cognition in mice transgenic for exon 1 of the human Huntington's disease mutation
K P Murphy, R J Carter, L A Lione, et al.
Genetic Analysis, Techniques and Applications
|
January 1, 1993
Preimplantation embryo sexing by polymerase chain reaction amplification of the sry gene on single mouse blastomeres
L C Mulder, M G Sacco, L Mangiarini, et al.
Cell
|
August 8, 1997
Huntingtin-encoded polyglutamine expansions form amyloid-like protein aggregates in vitro and in vivo
E Scherzinger, R Lurz, M Turmaine, et al.
Cell
|
November 1, 1996
Exon 1 of the HD gene with an expanded CAG repeat is sufficient to cause a progressive neurological phenotype in transgenic mice
L Mangiarini, K Sathasivam, M Seller, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
May 30, 1998
Altered brain neurotransmitter receptors in transgenic mice expressing a portion of an abnormal human huntington disease gene
J H Cha, C M Kosinski, J A Kerner, et al.
Cell
|
August 8, 1997
Formation of neuronal intranuclear inclusions underlies the neurological dysfunction in mice transgenic for the HD mutation
S W Davies, M Turmaine, B A Cozens, et al.
Philosophical Transactions of the Royal Society of London. Series B, Biological Sciences
|
August 6, 1999
Altered neurotransmitter receptor expression in transgenic mouse models of Huntington's disease
J H Cha, A S Frey, S A Alsdorf, et al.
Page
of 3
Search research articles
Search
Showing results (21-30 of 29) with videos related to
Sort By:
Page
of 3
You have reached the last page of results.
This site can display upto 29 results.
Neuroscience
|
October 19, 2000
Amyloid-like inclusions in Huntington's disease
D P McGowan, W van Roon-Mom, H Holloway, et al.
Human Molecular Genetics
|
April 10, 1999
Formation of polyglutamine inclusions in non-CNS tissue
K Sathasivam, C Hobbs, M Turmaine, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience
|
June 24, 2000
Abnormal synaptic plasticity and impaired spatial cognition in mice transgenic for exon 1 of the human Huntington's disease mutation
K P Murphy, R J Carter, L A Lione, et al.
Genetic Analysis, Techniques and Applications
|
January 1, 1993
Preimplantation embryo sexing by polymerase chain reaction amplification of the sry gene on single mouse blastomeres
L C Mulder, M G Sacco, L Mangiarini, et al.
Cell
|
August 8, 1997
Huntingtin-encoded polyglutamine expansions form amyloid-like protein aggregates in vitro and in vivo
E Scherzinger, R Lurz, M Turmaine, et al.
Cell
|
November 1, 1996
Exon 1 of the HD gene with an expanded CAG repeat is sufficient to cause a progressive neurological phenotype in transgenic mice
L Mangiarini, K Sathasivam, M Seller, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
May 30, 1998
Altered brain neurotransmitter receptors in transgenic mice expressing a portion of an abnormal human huntington disease gene
J H Cha, C M Kosinski, J A Kerner, et al.
Cell
|
August 8, 1997
Formation of neuronal intranuclear inclusions underlies the neurological dysfunction in mice transgenic for the HD mutation
S W Davies, M Turmaine, B A Cozens, et al.
Philosophical Transactions of the Royal Society of London. Series B, Biological Sciences
|
August 6, 1999
Altered neurotransmitter receptor expression in transgenic mouse models of Huntington's disease
J H Cha, A S Frey, S A Alsdorf, et al.
Page
of 3