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L Margolis

Showing results (591-600 of 862) with videos related to

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Developmental Cell|August 16, 2003
The mammalian passenger protein TD-60 is an RCC1 family member with an essential role in prometaphase to metaphase progressionCristiana Mollinari, Caroline Reynaud, Stephanie Martineau-Thuillier, et al.
Neurology|March 27, 2002
CAG/CTG repeat expansions at the Huntington's disease-like 2 locus are rare in Huntington's disease patientsG Stevanin, A Camuzat, S E Holmes, et al.
The Journal of Pediatrics|September 6, 2014
Initiation of oral contraceptives and changes in blood pressure and body mass index in healthy adolescentsElyse Olshen Kharbanda, Emily D Parker, Alan R Sinaiko, et al.
The Journal of Nervous and Mental Disease|February 2, 2016
Perspectives on the Use of eHealth in the Management of Patients With SchizophreniaGlenn J Treisman, Geetha Jayaram, Russell L Margolis, et al.
Schizophrenia Research|November 25, 2020
Better functional capacity and cognitive performance in clozapine responders compared to non-responders: A cross-sectional studyFrederick C Nucifora, Krista K Baker, Ashley Stricklin, et al.
Annals of Neurology|September 18, 2001
A disorder similar to Huntington's disease is associated with a novel CAG repeat expansionR L Margolis, E O'Hearn, A Rosenblatt, et al.
Annals of Neurology|February 28, 2012
Loss of junctophilin-3 contributes to Huntington disease-like 2 pathogenesisAna I Seixas, Susan E Holmes, Hiroshi Takeshima, et al.
Hypertension (Dallas, Tex. : 1979)|February 23, 2011
Missed opportunities in cardiovascular disease prevention?: low rates of hypertension recognition for women at medicine and obstetrics-gynecology clinicsJulie Schmittdiel, Joe V Selby, Bix Swain, et al.
Philosophical Transactions of the Royal Society of London. Series B, Biological Sciences|August 6, 1999
Polyglutamine pathogenesisC A Ross, J D Wood, G Schilling, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 15, 2001
The Gln-Ala repeat transcriptional activator CA150 interacts with huntingtin: neuropathologic and genetic evidence for a role in Huntington's disease pathogenesisS Holbert, I Denghien, T Kiechle, et al.
Pageof 87

Showing results (591-600 of 862) with videos related to

Sort By:
Pageof 87
Developmental Cell|August 16, 2003
The mammalian passenger protein TD-60 is an RCC1 family member with an essential role in prometaphase to metaphase progressionCristiana Mollinari, Caroline Reynaud, Stephanie Martineau-Thuillier, et al.
Neurology|March 27, 2002
CAG/CTG repeat expansions at the Huntington's disease-like 2 locus are rare in Huntington's disease patientsG Stevanin, A Camuzat, S E Holmes, et al.
The Journal of Pediatrics|September 6, 2014
Initiation of oral contraceptives and changes in blood pressure and body mass index in healthy adolescentsElyse Olshen Kharbanda, Emily D Parker, Alan R Sinaiko, et al.
The Journal of Nervous and Mental Disease|February 2, 2016
Perspectives on the Use of eHealth in the Management of Patients With SchizophreniaGlenn J Treisman, Geetha Jayaram, Russell L Margolis, et al.
Schizophrenia Research|November 25, 2020
Better functional capacity and cognitive performance in clozapine responders compared to non-responders: A cross-sectional studyFrederick C Nucifora, Krista K Baker, Ashley Stricklin, et al.
Annals of Neurology|September 18, 2001
A disorder similar to Huntington's disease is associated with a novel CAG repeat expansionR L Margolis, E O'Hearn, A Rosenblatt, et al.
Annals of Neurology|February 28, 2012
Loss of junctophilin-3 contributes to Huntington disease-like 2 pathogenesisAna I Seixas, Susan E Holmes, Hiroshi Takeshima, et al.
Hypertension (Dallas, Tex. : 1979)|February 23, 2011
Missed opportunities in cardiovascular disease prevention?: low rates of hypertension recognition for women at medicine and obstetrics-gynecology clinicsJulie Schmittdiel, Joe V Selby, Bix Swain, et al.
Philosophical Transactions of the Royal Society of London. Series B, Biological Sciences|August 6, 1999
Polyglutamine pathogenesisC A Ross, J D Wood, G Schilling, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 15, 2001
The Gln-Ala repeat transcriptional activator CA150 interacts with huntingtin: neuropathologic and genetic evidence for a role in Huntington's disease pathogenesisS Holbert, I Denghien, T Kiechle, et al.
Pageof 87