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Pflugers Archiv : European Journal of Physiology|July 1, 1996
Mechanisms responsible for quantal Ca2+ release from inositol trisphosphate-sensitive calcium storesJ B Parys, L Missiaen, H D Smedt, et al.
Cellular and Molecular Life Sciences : CMLS|October 19, 2012
Polycystins and cellular Ca2+ signalingD Mekahli, Jan B Parys, G Bultynck, et al.
The Journal of Biological Chemistry|February 25, 1994
Kinetics of empty store-activated Ca2+ influx in HeLa cellsL Missiaen, H De Smedt, J B Pary, et al.
The Biochemical Journal|May 15, 1994
Inhibition of inositol trisphosphate-induced calcium release by caffeine is prevented by ATPL Missiaen, J B Parys, H De Smedt, et al.
Gynecologic and Obstetric Investigation|January 1, 1990
Uterine norepinephrine levels are correlated with contraction force, but not with the occurrence of preeclampsiaG G Donders, L Missiaen, F P Zuspan, et al.
Molecular and Cellular Endocrinology|January 1, 1994
Partial calcium release in response to submaximal inositol 1,4,5-trisphosphate receptor activationL Missiaen, J B Parys, H De Smedt, et al.
The Biochemical Journal|January 1, 1991
Changes in the mechanism of Ca2(+) mobilization during the differentiation of BC3H1 muscle cellsH De Smedt, J B Parys, B Himpens, et al.
Pflugers Archiv : European Journal of Physiology|September 1, 1993
Bell-shaped activation of inositol-1,4,5-trisphosphate-induced Ca2+ release by thimerosal in permeabilized A7r5 smooth-muscle cellsJ B Parys, L Missiaen, H De Smedt, et al.
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