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Vestnik Rossiiskoi Akademii Meditsinskikh Nauk|December 18, 2007
[Progress in understanding moleculr mechanisms of oncogenesis, and novel methods of tumor growth control]L S Agapova, B P KopninCell Biology International|December 2, 1999
p53 does not control the spindle assembly cell cycle checkpoint but mediates G1 arrest in response to disruption of microtubule systemA A Sablina, L S Agapova, P M Chumakov, et al.Genetika|August 1, 1996
[Induction of hyperdiploidy and chromosome breaks in LIM1215 cells expressing the exogenous mutant p53]L S Agapova, N A Turovets, A V Ivanov, et al.Cancer Letters|December 19, 2008
p53 hot-spot mutants increase tumor vascularization via ROS-mediated activation of the HIF1/VEGF-A pathwayN V Khromova, P B Kopnin, E V Stepanova, et al.Genetika|April 26, 2000
[Effect of inactivating the p33ING1 tumor suppressor on the function of cell cycle "checkpoints" and genome stability]N A Turovets, L S Agapova, P B Kopnin, et al.Mutation Research|July 5, 1996
Chromosome changes caused by alterations of p53 expressionL S Agapova, G V Ilyinskaya, N A Turovets, et al.Journal of Cell Science|May 20, 1998
Activation of p53-mediated cell cycle checkpoint in response to micronuclei formationA A Sablina, G V Ilyinskaya, S N Rubtsova, et al.Molekuliarnaia Biologiia|April 29, 2005
[Retroviral reporter systems for the assessment of activity of stress-induced signal transduction pathways controlled by p53, HIF-1 and HSF-1 transcription factors]O V Razorenova, L S Agapova, A V Budanov, et al.Oncogene|May 26, 1999
P53-dependent effects of RAS oncogene on chromosome stability and cell cycle checkpointsL S Agapova, A V Ivanov, A A Sablina, et al.Molekuliarnaia Biologiia|August 10, 2007
[Transcriptional inhibition of human papilloma virus in cervical carcinoma cells reactivates functions of the tumor suppressor p53]D V Kochetkov, G V Il'inskaia, P G Komarov, et al.Pageof 2