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Reviews in the Neurosciences|April 1, 1995
Transgenic mice and modeling Alzheimer's diseaseL S Higgins, B CordellNeurodegeneration : a Journal for Neurodegenerative Disorders, Neuroprotection, and Neuroregeneration|June 1, 1995
Genetically engineered animal models of human neurodegenerative diseasesL S Higgins, B CordellAnnals of the New York Academy of Sciences|September 24, 1993
Transgenic mice expressing human beta-APP751, but not mice expressing beta-APP695, display early Alzheimer's disease-like histopathologyL S Higgins, R Catalano, D Quon, et al.The Journal of Biological Chemistry|April 22, 1994
Increased amyloid production from aberrant beta-amyloid precursor proteinsZ Zhong, D Quon, L S Higgins, et al.Proceedings of the National Academy of Sciences of the United States of America|June 6, 1995
Age-related learning deficits in transgenic mice expressing the 751-amino acid isoform of human beta-amyloid precursor proteinP M Moran, L S Higgins, B Cordell, et al.Proceedings of the National Academy of Sciences of the United States of America|May 9, 1995
Early Alzheimer disease-like histopathology increases in frequency with age in mice transgenic for beta-APP751L S Higgins, J M Rodems, R Catalano, et al.Annals of Neurology|May 1, 1994
Transgenic mouse brain histopathology resembles early Alzheimer's diseaseL S Higgins, D M Holtzman, J Rabin, et al.The American Journal of Pathology|August 1, 1996
P3 beta-amyloid peptide has a unique and potentially pathogenic immunohistochemical profile in Alzheimer's disease brainL S Higgins, G M Murphy, L S Forno, et al.Proceedings of the National Academy of Sciences of the United States of America|November 29, 2001
Specific spatial learning deficits become severe with age in beta -amyloid precursor protein transgenic mice that harbor diffuse beta -amyloid deposits but do not form plaquesM Koistinaho, M Ort, J M Cimadevilla, et al.Pageof 10