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L S Wicker

Showing results (21-30 of 56) with videos related to

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Journal of Autoimmunity|October 1, 1992
Acquired allo-tolerance to major or minor histocompatibility antigens indifferently contributes to preventing diabetes development in non-obese diabetic (NOD) miceC Carnaud, B Legrand, M Olivi, et al.
Journal of Immunology (Baltimore, Md. : 1950)|December 15, 1989
In vivo expression of perforin by CD8+ lymphocytes in autoimmune disease. Studies on spontaneous and adoptively transferred diabetes in nonobese diabetic miceL H Young, L B Peterson, L S Wicker, et al.
Journal of Immunology (Baltimore, Md. : 1950)|May 1, 1988
Activation of murine natural killer cells and macrophages by 8-bromoguanosineG C Koo, M E Jewell, C L Manyak, et al.
Diabetes|September 2, 2000
Analysis of the mouse CD30 gene: a candidate for the NOD mouse type 1 diabetes locus Idd9.2T Siegmund, N Armitage, L S Wicker, et al.
Diabetes|October 4, 2000
NOD Idd5 locus controls insulitis and diabetes and overlaps the orthologous CTLA4/IDDM12 and NRAMP1 loci in humansN J Hill, P A Lyons, N Armitage, et al.
The Journal of Experimental Medicine|September 1, 1993
I-E+ nonobese diabetic mice develop insulitis and diabetesP L Podolin, A Pressey, N H DeLarato, et al.
European Journal of Immunology|October 11, 1990
Suppression of B cell activation by cyclosporin A, FK506 and rapamycinL S Wicker, R C Boltz, V Matt, et al.
Diabetologia|March 6, 2007
A type 1 diabetes subgroup with a female bias is characterised by failure in tolerance to thyroid peroxidase at an early age and a strong association with the cytotoxic T-lymphocyte-associated antigen-4 geneJ M M Howson, D B Dunger, S Nutland, et al.
Journal of Autoimmunity|October 1, 1994
Responses of NOD congenic mice to a glutamic acid decarboxylase-derived peptideS L Chen, P J Whiteley, D C Freed, et al.
Cellular Immunology|May 1, 1987
Large, activated B cells are the primary B-cell target of 8-bromoguanosine and 8-mercaptoguanosineL S Wicker, R C Boltz, E A Nichols, et al.
Pageof 6

Showing results (21-30 of 56) with videos related to

Sort By:
Pageof 6
Journal of Autoimmunity|October 1, 1992
Acquired allo-tolerance to major or minor histocompatibility antigens indifferently contributes to preventing diabetes development in non-obese diabetic (NOD) miceC Carnaud, B Legrand, M Olivi, et al.
Journal of Immunology (Baltimore, Md. : 1950)|December 15, 1989
In vivo expression of perforin by CD8+ lymphocytes in autoimmune disease. Studies on spontaneous and adoptively transferred diabetes in nonobese diabetic miceL H Young, L B Peterson, L S Wicker, et al.
Journal of Immunology (Baltimore, Md. : 1950)|May 1, 1988
Activation of murine natural killer cells and macrophages by 8-bromoguanosineG C Koo, M E Jewell, C L Manyak, et al.
Diabetes|September 2, 2000
Analysis of the mouse CD30 gene: a candidate for the NOD mouse type 1 diabetes locus Idd9.2T Siegmund, N Armitage, L S Wicker, et al.
Diabetes|October 4, 2000
NOD Idd5 locus controls insulitis and diabetes and overlaps the orthologous CTLA4/IDDM12 and NRAMP1 loci in humansN J Hill, P A Lyons, N Armitage, et al.
The Journal of Experimental Medicine|September 1, 1993
I-E+ nonobese diabetic mice develop insulitis and diabetesP L Podolin, A Pressey, N H DeLarato, et al.
European Journal of Immunology|October 11, 1990
Suppression of B cell activation by cyclosporin A, FK506 and rapamycinL S Wicker, R C Boltz, V Matt, et al.
Diabetologia|March 6, 2007
A type 1 diabetes subgroup with a female bias is characterised by failure in tolerance to thyroid peroxidase at an early age and a strong association with the cytotoxic T-lymphocyte-associated antigen-4 geneJ M M Howson, D B Dunger, S Nutland, et al.
Journal of Autoimmunity|October 1, 1994
Responses of NOD congenic mice to a glutamic acid decarboxylase-derived peptideS L Chen, P J Whiteley, D C Freed, et al.
Cellular Immunology|May 1, 1987
Large, activated B cells are the primary B-cell target of 8-bromoguanosine and 8-mercaptoguanosineL S Wicker, R C Boltz, E A Nichols, et al.
Pageof 6