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Oncogene|April 21, 2001
p53-dependent radiation-induced crypt intestinal epithelial cells apoptosis is mediated in part through TNF-TNFR1 systemK Inagaki-Ohara, S Yada, N Takamura, et al.The Journal of Clinical Investigation|August 1, 1990
Elevated von Willebrand factor antigen is an early plasma predictor of acute lung injury in nonpulmonary sepsis syndromeD B Rubin, J P Wiener-Kronish, J F Murray, et al.Cell Death and Differentiation|May 30, 2015
Characterization of RIPK3-mediated phosphorylation of the activation loop of MLKL during necroptosisD A Rodriguez, R Weinlich, S Brown, et al.The Journal of Cell Biology|November 24, 1999
The pro-apoptotic proteins, Bid and Bax, cause a limited permeabilization of the mitochondrial outer membrane that is enhanced by cytosolR M Kluck, M D Esposti, G Perkins, et al.The Biochemical Journal|January 1, 1992
Purification and analysis of proteinase-resistant mutants of recombinant platelet-derived growth factor-BB exhibiting improved biological activityA L Cook, P M Kirwin, S Craig, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|August 15, 1997
Establishment of a cell-free system of neuronal apoptosis: comparison of premitochondrial, mitochondrial, and postmitochondrial phasesH M Ellerby, S J Martin, L M Ellerby, et al.Diabetes|June 1, 1988
Role of gluconeogenesis in sustaining glucose production during hypoglycemia caused by continuous insulin infusion in conscious dogsR T Frizzell, G K Hendrick, D W Biggers, et al.The EMBO Journal|May 15, 1996
The cytotoxic cell protease granzyme B initiates apoptosis in a cell-free system by proteolytic processing and activation of the ICE/CED-3 family protease, CPP32, via a novel two-step mechanismS J Martin, G P Amarante-Mendes, L Shi, et al.The Journal of Cell Biology|January 29, 1999
Ordering the cytochrome c-initiated caspase cascade: hierarchical activation of caspases-2, -3, -6, -7, -8, and -10 in a caspase-9-dependent mannerE A Slee, M T Harte, R M Kluck, et al.Cell Death and Differentiation|January 19, 2008
Blocking granule-mediated death by primary human NK cells requires both protection of mitochondria and inhibition of caspase activityK A Sedelies, A Ciccone, C J P Clarke, et al.Pageof 25