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Annals of the Rheumatic Diseases|August 1, 1979
Relationship between erythrocyte sedimentation rate and serum C-reactive protein in rheumatoid arthritisL Walsh, P Davies, B McConkeyBritish Medical Journal|February 16, 1980
Sulphasalazine in rheumatoid arthritisB McConkey, R S Amos, S Durham, et al.Current Opinion in Rheumatology|June 1, 1991
Disease-modifying antirheumatic drugs: gold, penicillamine, antimalarials, and sulfasalazineB McConkeyBritish Journal of Rheumatology|April 1, 1990
It is said that elevated erythrocyte sedimentation rate and the elevation of C-reactive protein (CRP) may be indicators of continuing joint destruction in rheumatoid arthritis. What then is the explanation for joint destruction in some patients in whom there is no such apparent elevation of either the sedimentation rate of CRP?B McConkeyAgents and Actions|June 1, 1978
Salazopyrin in rheumatoid arthritisB McConkey, R S Amos, E P Butler, et al.The Quarterly Journal of Medicine|January 1, 1986
The effect of antirheumatic drugs on circulating immune complexes in rheumatoid arthritisP J Forster, B McConkeyThe Journal of Rheumatology|December 1, 1984
Rheumatoid arthritis: the effects of treatment with dapsone on hemoglobinK A Grindulis, B McConkeyAnnals of the Rheumatic Diseases|June 1, 1984
Outcome of attempts to treat rheumatoid arthritis with gold, penicillamine, sulphasalazine, or dapsoneK A Grindulis, B McConkeyThe Journal of Rheumatology|October 1, 1990
Clinical and laboratory effects of prolonged therapy with sulfasalazine, gold or penicillamine: the effects of disease duration on treatment responseR D Situnayake, B McConkeyThe Journal of Rheumatology|January 1, 1988
Effects of rifampicin with and without isoniazid in rheumatoid arthritisB McConkey, R D SitunayakePageof 120