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Gastroenterology|April 1, 1993
Saccharomyces boulardii inhibits Clostridium difficile toxin A binding and enterotoxicity in rat ileumC Pothoulakis, C P Kelly, M A Joshi, et al.
Vaccine|October 1, 2016
Integrating pharmacies into public health program planning for pandemic influenza vaccine responseThomas J Fitzgerald, Yoonjae Kang, Carolyn B Bridges, et al.
Molecular & Cellular Proteomics : MCP|January 7, 2015
Identification of caveolar resident proteins in ventricular myocytes using a quantitative proteomic approach: dynamic changes in caveolar composition following adrenoceptor activationKrzysztof J Wypijewski, Michele Tinti, Wenzhang Chen, et al.
Veterinary Immunology and Immunopathology|January 5, 2021
Transcriptional response of blood leukocytes from turkeys challenged with Salmonella enterica serovar Typhimurium UK1Melissa S Monson, Bradley L Bearson, Matthew J Sylte, et al.
Methods and Protocols|October 26, 2021
Isolation of Live Leukocytes from Human Inflammatory MusclesJerome D Coudert, Emily McLeish, Anuradha Sooda, et al.
Molecular Oral Microbiology|October 10, 2018
Characterization and development of SAPP as a specific peptidic inhibitor that targets Porphyromonas gingivalisMeng-Hsuan Ho, Richard J Lamont, Walter J Chazin, et al.
Analytical and Bioanalytical Chemistry|March 26, 2021
Quantitative mass spectrometry imaging of drugs and metabolites: a multiplatform comparisonLieke Lamont, Darya Hadavi, Brent Viehmann, et al.
Journal of Geriatric Oncology|February 10, 2015
Comparing a medical records-based and a claims-based index for measuring comorbidity in patients with lung or colon cancerKenneth L Kehl, Elizabeth B Lamont, Barbara J McNeil, et al.
Journal of Medicinal Chemistry|December 1, 1981
Aporphines, 36. Dopamine receptor interactions of trihydroxyaporphines. Synthesis, radioreceptor binding, and striatal adenylate cyclase stimulation of 2,10,11-trihydroxyaporphines in comparison with other hydroxylated aporphinesJ L Neumeyer, G W Arana, S J Law, et al.
Brain Communications|June 27, 2025
Glycine-to-aspartic acid mutation at codon 51 in Snca disrupts the synaptic localisation of α-synuclein and enhances its propensity for synucleinopathyStephen West, Ammar Natalwala, Karamjit Singh Dolt, et al.
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