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Stem Cell Reports|August 1, 2020
Expression of Amyloidogenic Transthyretin Drives Hepatic Proteostasis Remodeling in an Induced Pluripotent Stem Cell Model of Systemic Amyloid DiseaseRichard M Giadone, Derek C Liberti, Taylor M Matte, et al.
Biorxiv : the Preprint Server for Biology|February 27, 2026
Pharmacological Inhibition of SLC33A1 Promotes Endoplasmic Reticulum Hyperoxidation and Induces Adaptive IRE1/XBP1s SignalingSergei Kutseikin, Maria Rafiq, Prerona Bora, et al.
Journal of the American Chemical Society|May 19, 2016
Arylfluorosulfates Inactivate Intracellular Lipid Binding Protein(s) through Chemoselective SuFEx Reaction with a Binding Site Tyr ResidueWentao Chen, Jiajia Dong, Lars Plate, et al.
Science Translational Medicine|September 15, 2017
Peptide probes detect misfolded transthyretin oligomers in plasma of hereditary amyloidosis patientsJoseph D Schonhoft, Cecilia Monteiro, Lars Plate, et al.
JCI Insight|April 10, 2020
Multiexon deletion alleles of ATF6 linked to achromatopsiaEun-Jin Lee, Wei-Chieh Jerry Chiang, Heike Kroeger, et al.
Biorxiv : the Preprint Server for Biology|August 2, 2024
Ribosomal Frameshifting Selectively Modulates the Assembly, Function, and Pharmacological Rescue of a Misfolded CFTR VariantPatrick Carmody, Francis J Roushar, Austin Tedman, et al.
Biorxiv : the Preprint Server for Biology|June 12, 2026
Profiling the CFTR Variant Selectivity and Off-Target Interactions of VX-121Ashish Rajesh Jhangiani, John A Olson, Austin Tedman, et al.
Proceedings of the National Academy of Sciences of the United States of America|October 10, 2024
Ribosomal frameshifting selectively modulates the assembly, function, and pharmacological rescue of a misfolded CFTR variantPatrick J Carmody, Francis J Roushar, Austin Tedman, et al.
Science Advances|October 29, 2025
Pharmacological targeting of RIG-I can selectively activate the integrated stress responseCaroline A Cuoco, Wen Ren, Kelsey R Baron, et al.
Hepatology (Baltimore, Md.)|April 9, 2025
Targeting the ER stress sensor IRE1 protects the liver from fibrosis through the downregulation of the proteostasis factor P4HB/PDIA1Younis Hazari, Lama Habbouche, Valeria A Garcia Lopez, et al.
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