Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Lauréline Deluche

Showing results (1-10 of 4) with videos related to

Pageof 1
Sort By:
Annales De Biologie Clinique|October 7, 2020
A curious chronic lymphocytic leukemiaInès Vergnolle, Lauréline Deluche
Blood|January 19, 2008
RUNX1 DNA-binding mutations and RUNX1-PRDM16 cryptic fusions in BCR-ABL+ leukemias are frequently associated with secondary trisomy 21 and may contribute to clonal evolution and imatinib resistanceCatherine Roche-Lestienne, Lauréline Deluche, Sélim Corm, et al.
Genes, Chromosomes & Cancer|September 4, 2008
Cryptic and partial deletions of PRDM16 and RUNX1 without t(1;21)(p36;q22) and/or RUNX1-PRDM16 fusion in a case of progressive chronic myeloid leukemia: a complex chromosomal rearrangement of underestimated frequency in disease progression?Lauréline Deluche, Sami Joha, Sélim Corm, et al.
The American Journal of Medicine|October 25, 2017
Effect of a Red Blood Cell Transfusion on Biological Markers Used to Determine the Cause of Anemia: A Prospective StudyAntoine Froissart, Benjamin Rossi, Brigitte Ranque, et al.
Pageof 1

Showing results (1-10 of 4) with videos related to

Sort By:
Pageof 1
Annales De Biologie Clinique|October 7, 2020
A curious chronic lymphocytic leukemiaInès Vergnolle, Lauréline Deluche
Blood|January 19, 2008
RUNX1 DNA-binding mutations and RUNX1-PRDM16 cryptic fusions in BCR-ABL+ leukemias are frequently associated with secondary trisomy 21 and may contribute to clonal evolution and imatinib resistanceCatherine Roche-Lestienne, Lauréline Deluche, Sélim Corm, et al.
Genes, Chromosomes & Cancer|September 4, 2008
Cryptic and partial deletions of PRDM16 and RUNX1 without t(1;21)(p36;q22) and/or RUNX1-PRDM16 fusion in a case of progressive chronic myeloid leukemia: a complex chromosomal rearrangement of underestimated frequency in disease progression?Lauréline Deluche, Sami Joha, Sélim Corm, et al.
The American Journal of Medicine|October 25, 2017
Effect of a Red Blood Cell Transfusion on Biological Markers Used to Determine the Cause of Anemia: A Prospective StudyAntoine Froissart, Benjamin Rossi, Brigitte Ranque, et al.
Pageof 1