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Journal of Neurochemistry|June 20, 2012
β-amyloid 1-42 induces physiological transcriptional regulation of BACE1Alessandra Piccini, Roberta Borghi, Michela Guglielmotto, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|March 11, 2011
Calcitonin gene-related peptide-mediated enhancement of purinergic neuron/glia communication by the algogenic factor bradykinin in mouse trigeminal ganglia from wild-type and R192Q Cav2.1 Knock-in mice: implications for basic mechanisms of migraine painStefania Ceruti, Giovanni Villa, Marta Fumagalli, et al.Neurobiology of Aging|February 4, 2014
Monomeric Aβ1-42 and RAGE: key players in neuronal differentiationSabrina Piras, Anna L Furfaro, Alessandra Piccini, et al.Biomacromolecules|January 22, 2016
Fate of PLA and PCL-Based Polymeric Nanocarriers in Cellular and Animal Models of Triple-Negative Breast CancerLeopoldo Sitia, Raffaele Ferrari, Martina B Violatto, et al.IJID Regions|April 3, 2025
Effectiveness and safety of tenofovir alafenamide/emtricitabine/bictegravir as a first-line regimen in people with HIV: A retrospective observational studyAndrea Giacomelli, Maria Vittoria Cossu, Davide Moschese, et al.The Journal of Biological Chemistry|January 31, 2013
An N-terminal fragment of the prion protein binds to amyloid-β oligomers and inhibits their neurotoxicity in vivoBrian R Fluharty, Emiliano Biasini, Matteo Stravalaci, et al.The Journal of Biological Chemistry|June 28, 2012
Specific recognition of biologically active amyloid-β oligomers by a new surface plasmon resonance-based immunoassay and an in vivo assay in Caenorhabditis elegansMatteo Stravalaci, Antonio Bastone, Marten Beeg, et al.International Journal of Molecular Sciences|September 23, 2022
Micro- and Nanoplastics' Effects on Protein Folding and AmyloidosisJoseph Windheim, Laura Colombo, Nora C Battajni, et al.Neurobiology of Disease|March 12, 2021
C. elegans detects toxicity of traumatic brain injury generated tauElisa R Zanier, Maria Monica Barzago, Gloria Vegliante, et al.Brain, Behavior, and Immunity|October 19, 2016
Toll-like receptor 4-dependent glial cell activation mediates the impairment in memory establishment induced by β-amyloid oligomers in an acute mouse model of Alzheimer's diseaseClaudia Balducci, Angelisa Frasca, Margherita Zotti, et al.Pageof 14