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Journal of Nuclear Medicine : Official Publication, Society of Nuclear Medicine|May 21, 2021
Glitter in the Darkness? Nonfibrillar β-Amyloid Plaque Components Significantly Impact the β-Amyloid PET Signal in Mouse Models of Alzheimer DiseaseGloria Biechele, Laura Sebastian Monasor, Karin Wind, et al.Neuroimage|July 28, 2024
Towards multicenter β-amyloid PET imaging in mouse models: A triple scanner head-to-head comparisonJohannes Gnörich, Mara Koehler, Karin Wind-Mark, et al.Elife|April 13, 2021
Microbiota-derived short chain fatty acids modulate microglia and promote Aβ plaque depositionAlessio Vittorio Colombo, Rebecca Katie Sadler, Gemma Llovera, et al.EMBO Molecular Medicine|May 25, 2019
Opposite microglial activation stages upon loss of PGRN or TREM2 result in reduced cerebral glucose metabolismJulia K Götzl, Matthias Brendel, Georg Werner, et al.Nature Neuroscience|January 9, 2019
Loss of TREM2 function increases amyloid seeding but reduces plaque-associated ApoESamira Parhizkar, Thomas Arzberger, Matthias Brendel, et al.Scientific Reports|July 29, 2018
Streptozotocin-induced β-cell damage, high fat diet, and metformin administration regulate Hes3 expression in the adult mouse brainPolyxeni Nikolakopoulou, Antonios Chatzigeorgiou, Ioannis Kourtzelis, et al.Science Translational Medicine|December 4, 2024
Myeloid cell-specific loss of NPC1 in mice recapitulates microgliosis and neurodegeneration in patients with Niemann-Pick type C diseaseLina Dinkel, Selina Hummel, Valerio Zenatti, et al.Pageof 2