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Cancers|September 9, 2022
Melanoma Tumour Vascularization and Tissue-Resident Endothelial Progenitor CellsGhazaleh Hashemi, James Dight, Kiarash Khosrotehrani, et al.
The British Journal of Dermatology|May 4, 2023
Impact of house dust mite in vitiligo skin: environmental contribution to increased cutaneous immunity and melanocyte detachmentHanene Bzioueche, Katia Boniface, Claire Drullion, et al.
The Journal of Investigative Dermatology|August 27, 2017
Melanocytes Sense Blue Light and Regulate Pigmentation through Opsin-3Claire Regazzetti, Laura Sormani, Delphine Debayle, et al.
The Journal of Investigative Dermatology|December 30, 2024
CLEC12B Regulates Melanocyte Immunity and Homeostasis in the Skin through the Signal Transducer and Activator of Transcription 1/IRF1 AxisLauriane Blot, Hanene Bzioueche, Marjorie Heim, et al.
Angiogenesis|May 11, 2024
Distinct roles of SOX9 in self-renewal of progenitors and mesenchymal transition of the endotheliumJilai Zhao, Laura Sormani, Sebastien Jacquelin, et al.
Nature Communications|May 8, 2021
Sox9 and Rbpj differentially regulate endothelial to mesenchymal transition and wound scarring in murine endovascular progenitorsJilai Zhao, Jatin Patel, Simranpreet Kaur, et al.
The Journal of Investigative Dermatology|July 26, 2021
CLEC12B Decreases Melanoma Proliferation by Repressing Signal Transducer and Activator of Transcription 3Henri Montaudié, Laura Sormani, Bérengère Dadone-Montaudié, et al.
The Journal of Investigative Dermatology|December 13, 2021
CLEC12B Is a Melanocytic Gene Regulating the Color of the SkinLaura Sormani, Henri Montaudie, Lauriane Blot, et al.
Nature Communications|May 18, 2019
Innate lymphocyte-induced CXCR3B-mediated melanocyte apoptosis is a potential initiator of T-cell autoreactivity in vitiligoMeri K Tulic, Elisa Cavazza, Yann Cheli, et al.
Cancer Cell|August 27, 2019
GAPDH Overexpression in the T Cell Lineage Promotes Angioimmunoblastic T Cell Lymphoma through an NF-κB-Dependent MechanismLaura Mondragón, Rana Mhaidly, Gian Marco De Donatis, et al.
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