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Cellular and Molecular Life Sciences : CMLS|July 21, 2016
Loss of mutL homolog-1 (MLH1) expression promotes acquisition of oncogenic and inhibitor-resistant point mutations in tyrosine kinasesLorraine Springuel, Elisabeth Losdyck, Pascale Saussoy, et al.
The Journal of Biological Chemistry|October 3, 2008
Ligand-independent homomeric and heteromeric complexes between interleukin-2 or -9 receptor subunits and the gamma chainYaniv Malka, Tekla Hornakova, Yohan Royer, et al.
The Journal of Biological Chemistry|January 14, 2009
Acute lymphoblastic leukemia-associated JAK1 mutants activate the Janus kinase/STAT pathway via interleukin-9 receptor alpha homodimersTekla Hornakova, Judith Staerk, Yohan Royer, et al.
Blood|February 20, 2010
ALL-associated JAK1 mutations confer hypersensitivity to the antiproliferative effect of type I interferonTekla Hornakova, Sabina Chiaretti, Muriel M Lemaire, et al.
Molecular Microbiology|November 27, 2007
The disulphide isomerase DsbC cooperates with the oxidase DsbA in a DsbD-independent mannerDidier Vertommen, Matthieu Depuydt, Jonathan Pan, et al.
Blood|October 30, 2014
Cooperating JAK1 and JAK3 mutants increase resistance to JAK inhibitorsLorraine Springuel, Tekla Hornakova, Elisabeth Losdyck, et al.
The Journal of Biological Chemistry|October 9, 2015
Distinct Acute Lymphoblastic Leukemia (ALL)-associated Janus Kinase 3 (JAK3) Mutants Exhibit Different Cytokine-Receptor Requirements and JAK Inhibitor SpecificitiesElisabeth Losdyck, Tekla Hornakova, Lorraine Springuel, et al.
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