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Bone|August 29, 2006
Low-dose estrogen treatment suppresses periosteal bone formation in response to mechanical loadingLeanne K Saxon, Charles H Turner
American Journal of Physiology. Endocrinology and Metabolism|December 1, 2005
Knee ligament mechanical properties are not influenced by estrogen or its receptorsStuart J Warden, Leanne K Saxon, Alesha B Castillo, et al.
Bone|March 23, 2011
Analysis of multiple bone responses to graded strains above functional levels, and to disuse, in mice in vivo show that the human Lrp5 G171V High Bone Mass mutation increases the osteogenic response to loading but that lack of Lrp5 activity reduces itLeanne K Saxon, Brendan F Jackson, Toshihiro Sugiyama, et al.
Journal of Biomechanics|July 19, 2005
Cellular accommodation and the response of bone to mechanical loadingJennifer L Schriefer, Stuart J Warden, Leanne K Saxon, et al.
Bone|June 10, 2008
Mechanical loading enhances the anabolic effects of intermittent parathyroid hormone (1-34) on trabecular and cortical bone in miceToshihiro Sugiyama, Leanne K Saxon, Gul Zaman, et al.
The Journal of Biological Chemistry|May 11, 2007
Wnt/beta-catenin signaling is a component of osteoblastic bone cell early responses to load-bearing and requires estrogen receptor alphaVictoria J Armstrong, Mariusz Muzylak, Andrew Sunters, et al.
Plos One|May 9, 2012
Lrp5 is not required for the proliferative response of osteoblasts to strain but regulates proliferation and apoptosis in a cell autonomous mannerBehzad Javaheri, Andrew Sunters, Gul Zaman, et al.
Bone|October 28, 2009
Loading-related regulation of gene expression in bone in the contexts of estrogen deficiency, lack of estrogen receptor alpha and disuseGul Zaman, Leanne K Saxon, Andrew Sunters, et al.
Bone|May 16, 2009
The mouse fibula as a suitable bone for the study of functional adaptation to mechanical loadingAlaa Moustafa, Toshihiro Sugiyama, Leanne K Saxon, et al.
The Journal of Biological Chemistry|November 4, 2011
Loading-related regulation of transcription factor EGR2/Krox-20 in bone cells is ERK1/2 protein-mediated and prostaglandin, Wnt signaling pathway-, and insulin-like growth factor-I axis-dependentGul Zaman, Andrew Sunters, Gabriel L Galea, et al.
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