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Lee Napierata

Showing results (1-10 of 6) with videos related to

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The European Journal of Neuroscience|July 1, 2008
Juvenile methylphenidate modulates reward-related behaviors and cerebral blood flow by decreasing cortical D3 receptorsSusan L Andersen, Lee Napierata, Heather C Brenhouse, et al.
Developmental Neuroscience|April 18, 2009
Juvenile methylphenidate exposure and factors that influence incentive processingHeather C Brenhouse, Lee Napierata, Lucinda Kussmaul, et al.
Arthritis and Rheumatism|January 30, 2010
Tumor necrosis factor alpha blockade exacerbates murine psoriasis-like disease by enhancing Th17 function and decreasing expansion of Treg cellsHak-Ling Ma, Lee Napierata, Nancy Stedman, et al.
The Journal of Investigative Dermatology|September 2, 2011
Inter-regulation of Th17 cytokines and the IL-36 cytokines in vitro and in vivo: implications in psoriasis pathogenesisYijun Carrier, Hak-Ling Ma, Hilda E Ramon, et al.
The Journal of Clinical Investigation|January 19, 2008
IL-22 is required for Th17 cell-mediated pathology in a mouse model of psoriasis-like skin inflammationHak-Ling Ma, Spencer Liang, Jing Li, et al.
Journal of Medicinal Chemistry|August 22, 2018
Discovery of 3-Cyano- N-(3-(1-isobutyrylpiperidin-4-yl)-1-methyl-4-(trifluoromethyl)-1 H-pyrrolo[2,3- b]pyridin-5-yl)benzamide: A Potent, Selective, and Orally Bioavailable Retinoic Acid Receptor-Related Orphan Receptor C2 Inverse AgonistMark E Schnute, Mattias Wennerstål, Jennifer Alley, et al.
Pageof 1

Showing results (1-10 of 6) with videos related to

Sort By:
Pageof 1
The European Journal of Neuroscience|July 1, 2008
Juvenile methylphenidate modulates reward-related behaviors and cerebral blood flow by decreasing cortical D3 receptorsSusan L Andersen, Lee Napierata, Heather C Brenhouse, et al.
Developmental Neuroscience|April 18, 2009
Juvenile methylphenidate exposure and factors that influence incentive processingHeather C Brenhouse, Lee Napierata, Lucinda Kussmaul, et al.
Arthritis and Rheumatism|January 30, 2010
Tumor necrosis factor alpha blockade exacerbates murine psoriasis-like disease by enhancing Th17 function and decreasing expansion of Treg cellsHak-Ling Ma, Lee Napierata, Nancy Stedman, et al.
The Journal of Investigative Dermatology|September 2, 2011
Inter-regulation of Th17 cytokines and the IL-36 cytokines in vitro and in vivo: implications in psoriasis pathogenesisYijun Carrier, Hak-Ling Ma, Hilda E Ramon, et al.
The Journal of Clinical Investigation|January 19, 2008
IL-22 is required for Th17 cell-mediated pathology in a mouse model of psoriasis-like skin inflammationHak-Ling Ma, Spencer Liang, Jing Li, et al.
Journal of Medicinal Chemistry|August 22, 2018
Discovery of 3-Cyano- N-(3-(1-isobutyrylpiperidin-4-yl)-1-methyl-4-(trifluoromethyl)-1 H-pyrrolo[2,3- b]pyridin-5-yl)benzamide: A Potent, Selective, and Orally Bioavailable Retinoic Acid Receptor-Related Orphan Receptor C2 Inverse AgonistMark E Schnute, Mattias Wennerstål, Jennifer Alley, et al.
Pageof 1