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Annals of Medicine|November 3, 2010
Tumor necrosis factor superfamily molecules in acute coronary syndromesPål Aukrust, Wiggo J Sandberg, Kari Otterdal, et al.
Mutation Research|June 2, 2009
Up-regulation of myocardial DNA base excision repair activities in experimental heart failureArne Yndestad, Christine G Neurauter, Erik Oie, et al.
Lipids in Health and Disease|December 31, 2011
Krill oil attenuates left ventricular dilatation after myocardial infarction in ratsLinn E Fosshaug, Rolf K Berge, Jan O Beitnes, et al.
Frontiers in Immunology|June 28, 2019
Mammalian Target of Rapamycin (mTOR) and the Proteasome Attenuates IL-1β Expression in Primary Mouse Cardiac FibroblastsMay-Kristin Torp, Kuan Yang, Trine Ranheim, et al.
American Journal of Physiology. Heart and Circulatory Physiology|December 28, 2010
Mechanisms of novel cardioprotective functions of CCN2/CTGF in myocardial ischemia-reperfusion injuryM Shakil Ahmed, Jørgen Gravning, Vladimir N Martinov, et al.
Journal of Cardiac Failure|August 2, 2015
Altered Levels of Fatty Acids and Inflammatory and Metabolic Mediators in Epicardial Adipose Tissue in Patients With Systolic Heart FailureLinn E Fosshaug, Christen P Dahl, Ivar Risnes, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 20, 2008
Uncovering G protein-coupled receptor kinase-5 as a histone deacetylase kinase in the nucleus of cardiomyocytesJeffrey S Martini, Philip Raake, Leif E Vinge, et al.
Journal of Applied Physiology (Bethesda, Md. : 1985)|February 21, 2012
Chemokines regulate small leucine-rich proteoglycans in the extracellular matrix of the pressure-overloaded right ventricleAnne Waehre, Maria Vistnes, Ivar Sjaastad, et al.
American Journal of Physiology. Heart and Circulatory Physiology|May 1, 2012
Cardiomyocyte-restricted inhibition of G protein-coupled receptor kinase-3 attenuates cardiac dysfunction after chronic pressure overloadThomas G von Lueder, Jørgen Gravning, Ole-Jakob How, et al.
Circulation|March 5, 2008
Targeted inhibition of cardiomyocyte Gi signaling enhances susceptibility to apoptotic cell death in response to ischemic stressBrent R DeGeorge, Erhe Gao, Matthieu Boucher, et al.
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