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Development (Cambridge, England)|February 24, 2006
Multiple roles of mesenchymal beta-catenin during murine limb patterningTheo P Hill, Makoto M Taketo, Walter Birchmeier, et al.Developmental Biology|March 18, 2008
Ca2+/Calmodulin-dependent kinase II signaling causes skeletal overgrowth and premature chondrocyte maturationMichael J Taschner, Mehran Rafigh, Fabienne Lampert, et al.Cancer Biotherapy & Radiopharmaceuticals|July 26, 2002
Treatment planning for molecular targeted radionuclide therapyChristine Hartmann Siantar, Kai Vetter, Gerald L DeNardo, et al.Medical Physics|March 6, 2004
Film dosimetry in the peripheral region using multiple sensitometric curvesRobin L Stern, John Kurylo, Christine Hartmann Siantar, et al.Frontiers in Cell and Developmental Biology|April 1, 2017
CaMKII Signaling Stimulates Mef2c Activity <i>In Vitro</i> but Only Minimally Affects Murine Long Bone Development <i>in vivo</i>Chandra S Amara, Christine Fabritius, Astrid Houben, et al.Developmental Cell|May 4, 2005
Canonical Wnt/beta-catenin signaling prevents osteoblasts from differentiating into chondrocytesTheo P Hill, Daniela Später, Makoto M Taketo, et al.Journal of Bone and Mineral Research : the Official Journal of the American Society for Bone and Mineral Research|May 13, 2022
Mice Lacking Wnt9a or Wnt4 Are Prone to Develop Spontaneous Osteoarthritis With Age and Display Alteration in Either the Trabecular or Cortical Bone CompartmentStefan Teufel, Lena Wolff, Ulrich König, et al.Nature Cell Biology|June 21, 2011
Differential requirement for the dual functions of β-catenin in embryonic stem cell self-renewal and germ layer formationNatalia Lyashenko, Markus Winter, Domenico Migliorini, et al.Developmental Dynamics : an Official Publication of the American Association of Anatomists|July 2, 2010
Mice lacking the orphan receptor ror1 have distinct skeletal abnormalities and are growth retardedNatalia Lyashenko, Martina Weissenböck, Amnon Sharir, et al.Development (Cambridge, England)|June 10, 2011
Ectodysplasin and Wnt pathways are required for salivary gland branching morphogenesisOtso Häärä, Sayumi Fujimori, Ruth Schmidt-Ullrich, et al.Pageof 6