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Lewis J Watson

Showing results (1-10 of 18) with videos related to

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Amino Acids|August 28, 2010
Augmented O-GlcNAc signaling attenuates oxidative stress and calcium overload in cardiomyocytesGladys A Ngoh, Lewis J Watson, Heberty T Facundo, et al.
American Journal of Physiology. Heart and Circulatory Physiology|August 30, 2011
Cardiac overexpression of 8-oxoguanine DNA glycosylase 1 protects mitochondrial DNA and reduces cardiac fibrosis following transaortic constrictionJianxun Wang, Qianwen Wang, Lewis J Watson, et al.
Journal of Molecular and Cellular Cardiology|June 10, 2008
Non-canonical glycosyltransferase modulates post-hypoxic cardiac myocyte death and mitochondrial permeability transitionGladys A Ngoh, Lewis J Watson, Heberty T Facundo, et al.
Advances in Physiology Education|August 8, 2024
Play your way to an "A": helping students engage during the social isolation of remote learningRobert E Brainard, Amy L Shaffer, Lewis J Watson, et al.
Plos One|January 12, 2013
Reduced cardiac fructose 2,6 bisphosphate increases hypertrophy and decreases glycolysis following aortic constrictionJianxun Wang, Jianxiang Xu, Qianwen Wang, et al.
American Journal of Physiology. Heart and Circulatory Physiology|July 18, 2006
Low-dose simvastatin improves survival and ventricular function via eNOS in congestive heart failureJames J M Greer, Aman K Kakkar, John W Elrod, et al.
American Journal of Physiology. Heart and Circulatory Physiology|May 6, 2008
Cardiac phosphatase-deficient 6-phosphofructo-2-kinase/fructose-2,6-bisphosphatase increases glycolysis, hypertrophy, and myocyte resistance to hypoxiaQianwen Wang, Rajakumar V Donthi, Jianxun Wang, et al.
American Journal of Physiology. Heart and Circulatory Physiology|March 13, 2012
O-GlcNAc signaling is essential for NFAT-mediated transcriptional reprogramming during cardiomyocyte hypertrophyHeberty T Facundo, Robert E Brainard, Lewis J Watson, et al.
Advances in Physiology Education|April 11, 2024
The Inverted Case Study Technique: changing the traditional case-study approach to prioritize physiological mechanisms over correct diagnosis and treatmentRobert E Brainard, Daniela G L Terson de Paleville, Michael C Long, et al.
The Journal of Biological Chemistry|September 4, 2014
MicroRNA-539 is up-regulated in failing heart, and suppresses O-GlcNAcase expressionSenthilkumar Muthusamy, Angelica M DeMartino, Lewis J Watson, et al.
Pageof 2

Showing results (1-10 of 18) with videos related to

Sort By:
Pageof 2
Amino Acids|August 28, 2010
Augmented O-GlcNAc signaling attenuates oxidative stress and calcium overload in cardiomyocytesGladys A Ngoh, Lewis J Watson, Heberty T Facundo, et al.
American Journal of Physiology. Heart and Circulatory Physiology|August 30, 2011
Cardiac overexpression of 8-oxoguanine DNA glycosylase 1 protects mitochondrial DNA and reduces cardiac fibrosis following transaortic constrictionJianxun Wang, Qianwen Wang, Lewis J Watson, et al.
Journal of Molecular and Cellular Cardiology|June 10, 2008
Non-canonical glycosyltransferase modulates post-hypoxic cardiac myocyte death and mitochondrial permeability transitionGladys A Ngoh, Lewis J Watson, Heberty T Facundo, et al.
Advances in Physiology Education|August 8, 2024
Play your way to an "A": helping students engage during the social isolation of remote learningRobert E Brainard, Amy L Shaffer, Lewis J Watson, et al.
Plos One|January 12, 2013
Reduced cardiac fructose 2,6 bisphosphate increases hypertrophy and decreases glycolysis following aortic constrictionJianxun Wang, Jianxiang Xu, Qianwen Wang, et al.
American Journal of Physiology. Heart and Circulatory Physiology|July 18, 2006
Low-dose simvastatin improves survival and ventricular function via eNOS in congestive heart failureJames J M Greer, Aman K Kakkar, John W Elrod, et al.
American Journal of Physiology. Heart and Circulatory Physiology|May 6, 2008
Cardiac phosphatase-deficient 6-phosphofructo-2-kinase/fructose-2,6-bisphosphatase increases glycolysis, hypertrophy, and myocyte resistance to hypoxiaQianwen Wang, Rajakumar V Donthi, Jianxun Wang, et al.
American Journal of Physiology. Heart and Circulatory Physiology|March 13, 2012
O-GlcNAc signaling is essential for NFAT-mediated transcriptional reprogramming during cardiomyocyte hypertrophyHeberty T Facundo, Robert E Brainard, Lewis J Watson, et al.
Advances in Physiology Education|April 11, 2024
The Inverted Case Study Technique: changing the traditional case-study approach to prioritize physiological mechanisms over correct diagnosis and treatmentRobert E Brainard, Daniela G L Terson de Paleville, Michael C Long, et al.
The Journal of Biological Chemistry|September 4, 2014
MicroRNA-539 is up-regulated in failing heart, and suppresses O-GlcNAcase expressionSenthilkumar Muthusamy, Angelica M DeMartino, Lewis J Watson, et al.
Pageof 2