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Plos One|April 12, 2012
Aberrant localization of FUS and TDP43 is associated with misfolding of SOD1 in amyotrophic lateral sclerosisEdward Pokrishevsky, Leslie I Grad, Masoud Yousefi, et al.
BMC Genomics|December 10, 2013
Kinotypes: stable species- and individual-specific profiles of cellular kinase activityBrett Trost, Jason Kindrachuk, Erin Scruten, et al.
Journal of Neuroinflammation|January 13, 2022
Sublethal enteroviral infection exacerbates disease progression in an ALS mouse modelYuan Chao Xue, Huitao Liu, Yasir Mohamud, et al.
The Journal of Infectious Diseases|August 1, 2009
Prion protein expression and release by mast cells after activationD James Haddon, Michael R Hughes, Frann Antignano, et al.
ACS Chemical Neuroscience|July 17, 2023
De Novo Design of a β-Helix Tau Protein Scaffold: An Oligomer-Selective Vaccine Immunogen Candidate for Alzheimer's DiseaseAdekunle Aina, Shawn C C Hsueh, Ebrima Gibbs, et al.
The Journal of Biological Chemistry|January 22, 2004
Monomeric Cu,Zn-superoxide dismutase is a common misfolding intermediate in the oxidation models of sporadic and familial amyotrophic lateral sclerosisRishi Rakhit, John P Crow, James R Lepock, et al.
Molecular Immunology|July 2, 2023
Distinct, age-dependent TLR7/8 signaling responses in porcine gamma-delta T cellsLeonie Bettin, Joseph Darbellay, Jill van Kessel, et al.
Veterinary Research|June 3, 2014
From mouth to macrophage: mechanisms of innate immune subversion by Mycobacterium avium subsp. paratuberculosisRyan J Arsenault, Pekka Maattanen, Joanna Daigle, et al.
Frontiers in Cellular Neuroscience|December 17, 2020
Amyotrophic Lateral Sclerosis: Proteins, Proteostasis, Prions, and PromisesLuke McAlary, Yee Lian Chew, Jeremy Stephen Lum, et al.
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