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Frontiers in Pharmacology|April 12, 2021
Insulin Resistance and Pellino-1 Mediated Decrease in the Activities of Vasodilator Signaling Contributes to Sunitinib-Induced HypertensionYang Liu, Liang-Liang Tang, Chen Liang, et al.Cellular Physiology and Biochemistry : International Journal of Experimental Cellular Physiology, Biochemistry, and Pharmacology|February 20, 2017
Transient Receptor Potential Melastatin 4 (TRPM4) Contributes to High Salt Diet-Mediated Early-Stage Endothelial InjuryXiao-Qing Ding, Tao Ban, Zeng-Yan Liu, et al.Journal of Hypertension|March 14, 2025
KAT7 contributes to ponatinib-induced hypertension by promoting endothelial senescence and inflammatory responses through activating NF-κB signaling pathwayLiang-Liang Tang, Xin-Yu Xu, Mei Zhang, et al.Oxidative Medicine and Cellular Longevity|November 16, 2020
Stimulation of Epithelial Sodium Channels in Endothelial Cells by Bone Morphogenetic Protein-4 Contributes to Salt-Sensitive Hypertension in RatsXu Yang, Na Niu, Chen Liang, et al.Biochimica Et Biophysica Acta. Molecular Basis of Disease|November 14, 2022
Inhibition of YAP by lenvatinib in endothelial cells increases blood pressure through ferroptosisChen Liang, Di Zhu, Wei Xia, et al.Journal of the American Heart Association|February 20, 2016
Angiotensin-Converting Enzyme 3 (ACE3) Protects Against Pressure Overload-Induced Cardiac HypertrophyChang-Jiang Yu, Liang-Liang Tang, Chen Liang, et al.Frontiers in Pharmacology|May 31, 2021
Cholesterol Stimulates the Transient Receptor Potential Melastatin 4 Channel in mpkCCDc14 CellsYong-Xu Cai, Bao-Long Zhang, Miao Yu, et al.Frontiers in Cell and Developmental Biology|July 5, 2021
Homocysteine Causes Endothelial Dysfunction via Inflammatory Factor-Mediated Activation of Epithelial Sodium Channel (ENaC)Chen Liang, Qiu-Shi Wang, Xu Yang, et al.Hepatology (Baltimore, Md.)|April 29, 2018
TRUSS Exacerbates NAFLD Development by Promoting IκBα Degradation in MiceChang-Jiang Yu, Qiu-Shi Wang, Ming-Ming Wu, et al.Frontiers in Pharmacology|October 24, 2022
Aldosterone-stimulated endothelial epithelial sodium channel (EnNaC) plays a role in cold exposure-induced hypertension in ratsLiang-Liang Tang, Xu Yang, Shu-Qi Yu, et al.Pageof 2