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Journal of Neurochemistry|July 1, 2018
A motor neuron strategy to save time and energy in neurodegeneration: adaptive protein stoichiometryElisabetta Zucchi, Ching-Hua Lu, Yunju Cho, et al.
Human Molecular Genetics|October 3, 2014
Expression of a pathogenic mutation of SOD1 sensitizes aprataxin-deficient cells and mice to oxidative stress and triggers hallmarks of premature ageingJean Carroll, Tristan K W Page, Shih-Chieh Chiang, et al.
Plos One|January 28, 2014
A nonsense mutation in mouse Tardbp affects TDP43 alternative splicing activity and causes limb-clasping and body tone defectsThomas Ricketts, Philip McGoldrick, Pietro Fratta, et al.
Molecular Neurodegeneration|May 4, 2023
Opinion: more mouse models and more translation needed for ALSElizabeth M C Fisher, Linda Greensmith, Andrea Malaspina, et al.
JCSM Rapid Communications|February 17, 2022
Ageing contributes to phenotype transition in a mouse model of periodic paralysisKaren J Suetterlin, S Veronica Tan, Roope Mannikko, et al.
Brain : a Journal of Neurology|August 2, 2019
Widespread FUS mislocalization is a molecular hallmark of amyotrophic lateral sclerosisGiulia E Tyzack, Raphaelle Luisier, Doaa M Taha, et al.
Plos One|March 12, 2010
Modification of superoxide dismutase 1 (SOD1) properties by a GFP tag--implications for research into amyotrophic lateral sclerosis (ALS)James C Stevens, Ruth Chia, William T Hendriks, et al.
Neurobiology of Aging|January 28, 2014
Widespread RNA metabolism impairment in sporadic inclusion body myositis TDP43-proteinopathyAndrea Cortese, Vincent Plagnol, Stefen Brady, et al.
Disease Models & Mechanisms|May 5, 2011
A comprehensive assessment of the SOD1G93A low-copy transgenic mouse, which models human amyotrophic lateral sclerosisAbraham Acevedo-Arozena, Bernadett Kalmar, Shafa Essa, et al.
Molecular Neurodegeneration|November 9, 2018
Tissue-enhanced plasma proteomic analysis for disease stratification in amyotrophic lateral sclerosisIrene Zubiri, Vittoria Lombardi, Michael Bremang, et al.
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