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Cancer Cell|October 6, 2009
Proteomic and genetic approaches identify Syk as an AML targetCynthia K Hahn, Jacob E Berchuck, Kenneth N Ross, et al.The Journal of Clinical Investigation|February 14, 2012
The intersection of genetic and chemical genomic screens identifies GSK-3α as a target in human acute myeloid leukemiaVersha Banerji, Stacey M Frumm, Kenneth N Ross, et al.Cell Death & Disease|June 17, 2022
Identification of an Epi-metabolic dependency on EHMT2/G9a in T-cell acute lymphoblastic leukemiaAnna Montanaro, Samuel Kitara, Elisa Cerretani, et al.Cancer Cell|June 15, 2021
STAG2 loss rewires oncogenic and developmental programs to promote metastasis in Ewing sarcomaBiniam Adane, Gabriela Alexe, Bo Kyung A Seong, et al.Science Translational Medicine|March 9, 2018
Exploiting an Asp-Glu "switch" in glycogen synthase kinase 3 to design paralog-selective inhibitors for use in acute myeloid leukemiaFlorence F Wagner, Lina Benajiba, Arthur J Campbell, et al.The Journal of Clinical Investigation|December 5, 2017
CRISPR-Cas9 screen reveals a MYCN-amplified neuroblastoma dependency on EZH2Liying Chen, Gabriela Alexe, Neekesh V Dharia, et al.Science Translational Medicine|April 1, 2021
Targeting acute myeloid leukemia dependency on VCP-mediated DNA repair through a selective second-generation small-molecule inhibitorBlandine Roux, Camille Vaganay, Jesse D Vargas, et al.ACS Chemical Biology|April 30, 2016
Inhibitors of Glycogen Synthase Kinase 3 with Exquisite Kinome-Wide Selectivity and Their Functional EffectsFlorence F Wagner, Joshua A Bishop, Jennifer P Gale, et al.Pageof 7