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Biorxiv : the Preprint Server for Biology|April 10, 2026
c-MYC is an aggregation-prone, amyloidogenic proteinLing Lin, Kun-Han Chuang, Chengkai DaiBiorxiv : the Preprint Server for Biology|March 16, 2026
Driving proteomic imbalance in malignancy provokes proteomic catastrophe and confers tumor suppressionBabul Moni Ram, Omprakash Shriwas, Meng Xu, et al.Cell Reports|May 24, 2023
Heat shock factor 1 (HSF1) specifically potentiates c-MYC-mediated transcription independently of the canonical heat shock responseMeng Xu, Ling Lin, Babul Moni Ram, et al.Philosophical Transactions of the Royal Society of London. Series B, Biological Sciences|December 6, 2017
The heat-shock, or HSF1-mediated proteotoxic stress, response in cancer: from proteomic stability to oncogenesisChengkai DaiMolecular & Cellular Oncology|March 15, 2016
Metabolic stressors disrupt proteome homeostasis to suppress malignancyChengkai DaiFuture Oncology (London, England)|March 25, 2006
HSP90: a rising star on the horizon of anticancer targetsChengkai Dai, Luke WhitesellMethods in Molecular Biology (Clifton, N.J.)|December 8, 2016
Visualization of RAS/MAPK Signaling In Situ by the Proximity Ligation Assay (PLA)Zijian Tang, Chengkai DaiBioessays : News and Reviews in Molecular, Cellular and Developmental Biology|March 16, 2017
mTORC1 senses stresses: Coupling stress to proteostasisKuo-Hui Su, Chengkai DaiCancer Journal (Sudbury, Mass.)|June 6, 2003
Astrocyte differentiation states and glioma formationChengkai Dai, Eric C HollandTrends in Cell Biology|November 25, 2015
HSF1: Guardian of Proteostasis in CancerChengkai Dai, Stephen Byers SampsonPageof 339