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The Journal of Experimental Medicine|April 25, 2007
Regulation of AID expression in the immune responseElizabeth E Crouch, Zhiyu Li, Makiko Takizawa, et al.
The Journal of Clinical Investigation|August 18, 2004
Poor immunogenicity of a self/tumor antigen derives from peptide-MHC-I instability and is independent of toleranceZhiya Yu, Marc R Theoret, Christopher E Touloukian, et al.
Molecular Carcinogenesis|February 13, 2013
Normal viability of Kai1/Cd82 deficient miceJohn I Risinger, Mary Custer, Lionel Feigenbaum, et al.
Cell Cycle (Georgetown, Tex.)|September 22, 2010
Epimorphic regeneration in mice is p53-independentL Matthew Arthur, Renee M Demarest, Lise Clark, et al.
Immunity|November 21, 2007
Deletion of CD4 and CD8 coreceptors permits generation of alphabetaT cells that recognize antigens independently of the MHCFrançois Van Laethem, Sophia D Sarafova, Jung-Hyun Park, et al.
Nature Immunology|April 8, 2015
Let-7 microRNAs target the lineage-specific transcription factor PLZF to regulate terminal NKT cell differentiation and effector functionLeonid A Pobezinsky, Ruth Etzensperger, Susanna Jeurling, et al.
The Journal of Cell Biology|August 8, 2012
Loss of ATM kinase activity leads to embryonic lethality in miceJeremy A Daniel, Manuela Pellegrini, Baeck-Seung Lee, et al.
Blood|March 10, 2012
Basis of CTLA-4 function in regulatory and conventional CD4(+) T cellsXuguang Tai, François Van Laethem, Leonid Pobezinsky, et al.
The Journal of Experimental Medicine|August 6, 2008
AID expression levels determine the extent of cMyc oncogenic translocations and the incidence of B cell tumor developmentMakiko Takizawa, Helena Tolarová, Zhiyu Li, et al.
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